Nocodazole

For research use only.

Catalog No.S2775 Synonyms: Oncodazole

35 publications

Nocodazole Chemical Structure

Molecular Weight(MW): 301.32

Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.

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Selleck's Nocodazole has been cited by 35 publications

Purity & Quality Control

Choose Selective Microtubule Associated Inhibitors

Biological Activity

Description Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.
Targets
Microtubules [2]
(Cell-free assay)
Abl [1]
(Cell-free assay)
Abl (E255K) [1]
(Cell-free assay)
Abl (T315I) [1]
(Cell-free assay)
0.21 μM 0.53 μM 0.64 μM
In vitro

Nocodazole is a high-affinity ligand for the cancer-related kinases including Abl phosphorylated, c-Kit, BRAF, and MEK with Kd of 0.091 μM, 1.6 μM, 1.8 μM and 1.6 μM, respectively. In addition, the Kd of Nocodazole for Abl(E255K) phosphorylated, Abl(T315I) phosphorylated, BRAF(V600E) and PI3Kγ is 0.12 μM, 0.17 μM, 1.1 μM and 1.5 μM, respectively. Nocodazole induces apoptosis in chronic lymphocytic leukemia cells. Nocodazole inhibits insulin-stimulated glucose transport. Nocodazole decreases apoptosis in some human colon carcinoma cells. Nocodazole impairs the morphology and directionality of migrating medial gan-glionic eminence cells. [1] At high concentrations, Nocodazole rapidly depolymerizes microtubules in cells, while low concentrations of Nocodazole inhibit microtubule dynamic instability. [2] Mitotic cells incubated with different concentrations of Paclitaxel are inhibited from progressing to G1 phase 6 hours after release from the Nocodazole block, with a median inhibitory concentration of 4 nM. Nocodazole-pretreated cells exposed to Paclitaxel in the absence of Nocodazole only form free-floating microtubules, whereas pretreated cells exposed to Paclitaxel in the presence of Nocodazole-assembled centrosome organize microtubules. [3] Nocodazole disrupts microtubules by binding to β-tubulin. Nocodazole prevents the formation of one of the two interchain disulfide linkages. Nocodazole impairs the transport of vesicles. Nocodazole suppress METH-induced cell death and lysosomal dysfunction. METH-induced cell death is significantly decreased by Nocodazole pretreatment in comparison to METH alone. [4]Nocodazole doubles HDR efficiency to up to 30% in iPSCs. It improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing[6].

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
rat LT12 cells MoHWVJJwdGmoZYLheIlwdiCjc4PhfS=> Ml;yOFghcA>? NVnz[5VoSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBz[XRiTGSxNkBk\WyuczDh[pRmeiB2ODDodpMh[nliWGTUJIF{e2G7LDDJR|UxRTZibl2= NUnoR2Q2OTl{MkCwNVg>
human PANC1 cells M4ft[XBzd2yrZnXyZZRqd25iYYPzZZk> MojQOFghcA>? NYnLUmNXSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDQRW5EOSClZXzsd{Bi\nSncjC0PEBpenNiYomgV3JDKGG|c3H5MEBIUTVyPUGwJI5O NET6c2wzPTh{N{WyNi=>
human HeLa cells M1jINXBzd2yrZnXyZZRqd25iYYPzZZk> M4H2bFQ5KGh? NFTtWlVCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIFjlUIEh[2WubIOgZYZ1\XJiNEigbJJ{KGK7IGPSRkBie3OjeTygS2k2OD1zNTDuUS=> NFzNcmEzPTh{N{WyNi=>
human MDA-MB-231 cells NXvMR5I4WHKxbHnm[ZJifGmxbjDhd5NigQ>? Mmr4OFghcA>? MWXBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKE2GQT3NRk0zOzFiY3XscJMh[W[2ZYKgOFghcHK|IHL5JHNTSiCjc4PhfUwhT0l3ME2xO{BvVQ>? MVqyOVgzPzV{Mh?=
human A549 cells NH;NTndRem:uaX\ldoF1cW:wIHHzd4F6 NHPTc3k1QCCq M4DEPGFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iQUW0PUBk\WyuczDh[pRmeiB2ODDodpMh[nliU2LCJIF{e2G7LDDHTVUxRTJyIH7N MorQNlU5Ojd3MkK=
lymphoid leukemia L1210 cells MU\Qdo9tcW[ncnH0bY9vKGG|c3H5 NG\PdpU1QCCq NWjHOYNzUW6qaXLpeI9zgSClb37j[Y51emG2aX;uJIFo[Wmwc4SgdJJwdGmoZYLheIlwdiCxZjDjeYx1fXKnZDDsfY1xcG:rZDDs[ZVs\W2rYTDMNVIyOCClZXzsd{BlfXKrbnegOFghcHK|LDDJSFUxRTJ5IH7N NHXHXY84OTNzNEiz
human A549 cells  MmnaR5l1d3SxeHnjxsBie3OjeR?= NH3TTXAzPCCq MYrDfZRwfG:6aXPpeJkh[WejaX7zeEBpfW2jbjDBOVQ6KGOnbHzzJIFnfGW{IEK0JIhzeyCkeTDNWHQh[XO|YYmsJGlEPTB;M{[uOkBvVQ>? MoDvNlAyOTBzM{e=
L1210 murine leukemia cell MnHrS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NIrmVJFKdiC4aYTyc{Bnd3JiaYTzJIlvcGmkaYTvdpkh[WO2aY\peJkh[WejaX7zeEBNOTJzMDDteZJqdmVibHX1b4VucWFiY3XscEBoem:5dHisJGlEPTB;M{igcm0> MYSxO|M5OTR4
mouse P388 cells M3TXZXBzd2yrZnXyZZRqd25iYYPzZZk> Mmm0OFghcA>? MYTBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IH3veZNmKFB|OEigZ4VtdHNiYX\0[ZIhPDhiaILzJIJ6KFiWVDDhd5NigSxiSVO1NF01OCCwTR?= M{T1b|E6OjJyMEG4
LT12 cells NFvsc49Rem:uaX\ldoF1cW:wIHHzd4F6 MYS0PEBp NYnoTIRYSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBNXDF{IHPlcIx{KGK7IGjUWEBie3OjeTDh[pRmeiB2ODDodpMtKEmFNUC9OFAhdk1? M4HlWFE4OThzMU[0
human U2OS cells NEDoSndHfW6ldHnvckBie3OjeR?= MXu3NkBp M4[wTmlv\HWldHnvckBw\iCvaYTveIlkKGG{cnXzeEBqdiCqdX3hckBWOk:VIHPlcIx{KGG|c3Xzd4VlKGG|IH3peI91cWNiaX7k[Zgh[W[2ZYKgO|IhcHK|IHL5JI1q[3Kxc3PvdIlkKGGwYXz5d4l{NCCHQ{WwQVUxKG6P M1v0c|I1QTByOEi3
human NCI-H460 cells M1joPXBzd2yrZnXyZZRqd25iYYPzZZk> MYC0PEBp MXfBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKE6FST3IOFYxKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD14ODDuUS=> MnLONlE2PjN5NUC=
human BT549 cells MUnQdo9tcW[ncnH0bY9vKGG|c3H5 NXvVcFBEPDhiaB?= NUHvUI1uSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDCWFU1QSClZXzsd{Bi\nSncjC0PEBpenNiYomgZYxidWG{IHLseYUh[XO|YYmsJGlEPTB;Nkmgcm0> NWTKbXJOOjF3NkO3OVA>
human 451LU cells NU\GOoRXWHKxbHnm[ZJifGmxbjDhd5NigQ>? MXq0PEBp NUmycJFLSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjC0OVFNXSClZXzsd{Bi\nSncjC0PEBpenNiYomgZYxidWG{IHLseYUh[XO|YYmsJGlEPTB;N{Kgcm0> M2jCblIyPTZ|N{Ww
human SKOV3 cells MV3Qdo9tcW[ncnH0bY9vKGG|c3H5 NFnS[Y81QCCq NVe1[3JtSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDTT29XOyClZXzsd{Bi\nSncjC0PEBpenNiYomgZYxidWG{IHLseYUh[XO|YYmsJGlEPTB;OECgcm0> MYKyNVU3Ozd3MB?=
K562 cells MX3GeY5kfGmxbjDhd5NigQ>? NWC0NnM6TW[oZXP0bZZmKGOxbnPlcpRz[XSrb36gdoVyfWm{ZXSgeI8h[XK{ZYP0JJBzd3CrZHn1cUBqd2SrZHWgd5RicW6nZDDLOVYzKGOnbHzzJIlvKEd{L12gdIhie2VuIFXDOVA:QDJibl2= MmHpNVI5PTJ5Nki=
human SKBR3 cells MkfiS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NX\LU2RIPDhiaB?= M1fQfmdzd3e2aDDpcohq[mm2aX;uJI9nKGi3bXHuJHNMSlJ|IHPlcIx{KGGodHXyJFQ5KGi{czDifUBOXFRiYYPzZZk> MXWyNlg2ODJzNB?=
human COLO205 cells MVHQdo9tcW[ncnH0bY9vKGG|c3H5 MkO2OFghcA>? MmXDRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCFT1zPNlA2KGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTCsJGlEPTB;OEigcm0> MkHGNlE2PjN5NUC=
human KB/HeLa cells NV[0VW9ZTnWwY4Tpc44h[XO|YYm= MWeyOEBp M4DKSmNmdGxiY4njcIUh[XK{ZYP0JIlvKGi3bXHuJGtDN0inTHGgZ4VtdHNiYYPz[ZN{\WRiYYOgS|IwVSCyaHHz[UBi[2O3bYXsZZRqd25iYX\0[ZIhOjRiaILzJIJ6KE[DQ2OgZY5idHm|aYOsJGVEPTB;OUGgcm0> NWPVeFR6OTl{MkCwNVg>
human A549 cells MUTHdo94fGhiaX7obYJqfGmxbjDhd5NigQ>? M{nRWGdzd3e2aDDpcohq[mm2aX;uJI9nKGi3bXHuJGE2PDliY3XscJMh[nliU4Xs[o9zcG:mYX3pcoUhSiCjc4PhfUwhT0l3ME2wMlEh|ryP MWOyNVExPjR3OB?=
human MCF7 cells MlK5S5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NXTZTXRWT3Kxd4ToJIlvcGmkaYTpc44hd2ZiaIXtZY4hVUOINzDj[YxteyCkeTDTeYxnd3Kqb3ThcYlv\SCEIHHzd4F6NCCJSUWwQVAvOSEQvF2= MUWyNVExPjR3OB?=
human RKO cells M2X1[HBzd2yrZnXyZZRqd25iYYPzZZk> MWjBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKFKNTzDj[YxteyCkeTDYWHQh[XO|YYmsJGlEPTB;MD6xNUDPxE1? MnTmNVkzOjByMUi=
human SW480 cells M4GyT3Bzd2yrZnXyZZRqd25iYYPzZZk> MWO0PEBp NVTueXc1SW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDTW|Q5OCClZXzsd{Bi\nSncjC0PEBpenNiYomgZYxidWG{IHLseYUh[XO|YYmsJGlEPTB;MD6xNkDPxE1? M2\i[lIyPTZ|N{Ww
human NCI-H460 cells MX\Qdo9tcW[ncnH0bY9vKGG|c3H5 MWi0PEBp NWnhT4hqSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDOR2kuUDR4MDDj[YxteyCjZoTldkA1QCCqcoOgZpkhYFSWIHHzd4F6NCCLQ{WwQVAvOTVizszN MojVNlA2Ozd5NkW=
human SF268 cells M{nQZmN6fG:2b4jpZ:Kh[XO|YYm= MlK4OFghcA>? M4jpcWN6fG:2b4jpZ4l1gSCjZ3HpcpN1KGi3bXHuJHNHOjZ6IHPlcIx{KGGodHXyJFQ5KGi{czDifUBZXFRiYYPzZZktKEmFNUC9NE4{KM7:TR?= MXGyNVcxPTJ{Mx?=
human SK-N-SH cells NULhZpJET3Kxd4ToJIlvcGmkaYTpc44h[XO|YYm= MmjXS5Jwf3SqIHnubIljcXSrb36gc4YhcHWvYX6gV2suVi2VSDDj[YxteyCkeTDTeYxnd3Kqb3ThcYlv\SCEIHHzd4F6NCCJSUWwQVAvPjZizszN MXiyNVExPjR3OB?=
human MIAPaCa2 cells Mkj6VJJwdGmoZYLheIlwdiCjc4PhfS=> M{PYSFQ5KGh? MUDBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKE2LQWDhR4EzKGOnbHzzJIF{e2W|c3XkJIF{KGe{b4f0bEBqdmirYnn0bY9vKGGodHXyJFQ5KGi{czDifUBUWkJiYYPzZZktKEeLNUC9NE46PSEQvF2= MV6yOVE{OTl3Nh?=
human HaCaT cells MVrDfZRwfG:6aXRCpIF{e2G7 MmDyOFghcA>? NX;NeJVUS3m2b4TvfIlkKGGldHn2bZR6KGGpYXnud5QhcHWvYX6gTIFE[VRiY3XscJMh[XO|ZYPz[YQh[XNicnXkeYN1cW:wIHnuJINmdGxiZ4Lve5RpKGmwY4XiZZRm\CCob4KgOFghcHK|IHL5JG1VXCCjc4PhfUwhUUN3ME24Mlkh|ryP NHLGWHAzPjJ|MUC4NC=>
human SGC7901 cells M3rxOGZ2dmO2aX;uJIF{e2G7 MmrvOFghcA>? MX;JcoR2[3Srb36gc4Yh\Gm|dH;yeIlwdnNiaX6geJVjfWyrbjDhd5NmdWKueTDpckBpfW2jbjDTS2M4QTBzIHPlcIx{KGG|c3Xzd4VlKGG|IHHico9zdWGuIH3peI91cWNic4DpcoRt\SCob4LtZZRqd25iYYSgNkBnd2ymIFnDOVAhdGW4ZXygbY5kfWKjdHXkJIZweiB2ODDodpMh[nliaX3teY5w\my3b4Lld4NmdmOnIIP0ZYlvcW6pIHLhd4VlKG[udX;y[ZNk\W6lZTDtbYNzd3Olb4D5 MVmyOVI3OjB3MR?=
human Jurkat T cells M3zsbWZ2dmO2aX;uJIF{e2G7 M2fQb|M3KGh? NWX4fWUxUW6mdXP0bY9vKG:oIH3pZ5JwfHWkdXzlJIRidWGpZTDpckBpfW2jbjDKeZJs[XRiVDDj[YxteyCxdnXy[ZhxemW|c3nu[{BD[2x{IHHmeIVzKDN4IHjyd{BjgSCrbX31co9{fGGrbnnu[{Bu\XSqb3S= NFL1fXYzOjF7N{O5Ny=>
human PC3M cells NWPtTXFTTnWwY4Tpc44h[XO|YYm= NW\C[Y1rOC53IIXnM41t M4DOZ|I1KGh? M4PjSmlv\HWldHnvckBw\iCjcH;weI9{cXNiaX6gbJVu[W5iUFOzUUBk\WyuczDhd5Nme3OnZDDhd{Bi[2O3bYXsZZRqd25iYYSgd5VjTzFicHjhd4Uh[XRiMD61JJVoN22uIHHmeIVzKDJ2IHjyd{BjgSCobH;3JIN6fG:vZYTyfS=> NUnzRWw4OTh{NEe1O|M>
mouse NIH3T3 cells NFz5b2VHfW6ldHnvckBie3OjeR?= NWO4PJlbOC53IIXnM41t NUTPTnp5OjRiaB?= NHficZZKdmS3Y4Tpc44hd2ZiYYDvdJRwe2m|IHnuJI1wfXOnIF7JTFNVOyClZXzsd{Bie3Onc4Pl[EBieyCjY3P1cZVt[XSrb36gZZQhe3WkR{GgdIhie2ViYYSgNE42KHWpL33sJIFnfGW{IEK0JIhzeyCkeTDmcI94KGO7dH;t[ZRzgQ>? NIHx[4QyQDJ2N{W3Ny=>

... Click to View More Cell Line Experimental Data

Assay
Methods Test Index PMID
Western blot
Cdc2 / cyclin B1 / MAD2 / Cdc20 ; 

PubMed: 21918689     


E (upper part). Time-dependent changes in cyclin B1 and Cdc2 protein levels following nocodazole treatment. Cells were treated with nocodazole (250 nM) for the length of time as indicated, and whole cell lysates were prepared. An equal amount of protein lysates was electrophoretically separated on the 10% SDS-polyacrylamide gel, and transferred to nitrocellulose membrane. Western blots were detected using specific antibodies against cyclin B1, Cdc2 (CDK1), MAD2, and Cdc20 on an enhanced chemiluminescence (ECL) apparatus. Membrane was stripped for determining the levels of GAPDH as a loading control. E (lower part). The relative protein levels for cyclin B1, Cdc2, MAD2, and Cdc20 were calculated according to their densitometry readings, which were normalized according to the corresponding readings for the GAPDH protein bands. Each value is mean ± S.D. from three replicate determinations. * P<0.05, ** P<0.01 versus vehicle-treated control. 

21918689
Immunofluorescence
Tubulin / LC8 ; 

PubMed: 23038268     


Shown is endogenous LC8 colocalized with microtubules in HeLa cells. Control, 20 nm taxol- and 100 nm nocodazole-treated cells were stained with α-tubulin and LC8 antibodies. Scale bar, 20 μm.

Brd4 / α-tubulin ; 

PubMed: 22567088     


Cells were treated with indicated concentrations of nocodazole for 4 h, and mitotic cells were immunostained for endogenous Brd4 and α-tubulin. DNA was stained by Hoechst33324.

23038268 22567088
Growth inhibition assay
Cell viability ; 

PubMed: 21918689     


Changes in cell viability (MTT assay) after treatment of cells with 250 nM nocodazole for different lengths of time. Each data point is the mean ± S.D. from four replicate measurements from one representative experiment. 

21918689
In vivo The tumor volume and tumor weight of the mice treated with Ketoconazole  plus  Nocodazole are significantly reduced as compared with those treated with Ketoconazole or Nocodazole alone. Combined treatment with Ketoconazole  plus  Nocodazole strongly enhances apoptosis of COLO 205 tumor xenografts treated with Ketoconazole  or  Nocodazole alone. [5]

Protocol

Animal Research:

[5]

- Collapse
  • Animal Models: Nude mice with COLO-205 tumor xenografts
  • Dosages: 5 mg/kg
  • Administration: Administered via i.p.
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 7 mg/mL (23.23 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents to the product individually and in order(Data is from Selleck tests instead of citations):
2% DMSO+30% PEG 300+2% Tween 80+ddH2O
For best results, use promptly after mixing.
1mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 301.32
Formula

C14H11N3O3S

CAS No. 31430-18-9
Storage powder
in solvent
Synonyms Oncodazole
Smiles COC(=O)NC1=NC2=CC=C(C=C2[NH]1)C(=O)C3=CC=CS3

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Microtubule Associated Signaling Pathway Map

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID