Nocodazole

Catalog No.S2775 Synonyms: Oncodazole

Nocodazole Chemical Structure

Molecular Weight(MW): 301.32

Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.

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Cited by 31 Publications

Purity & Quality Control

Choose Selective Microtubule Associated Inhibitors

Biological Activity

Description Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.
Targets
Microtubules [2]
(Cell-free assay)
Abl [1]
(Cell-free assay)
Abl (E255K) [1]
(Cell-free assay)
Abl (T315I) [1]
(Cell-free assay)
0.21 μM 0.53 μM 0.64 μM
In vitro

Nocodazole is a high-affinity ligand for the cancer-related kinases including Abl phosphorylated, c-Kit, BRAF, and MEK with Kd of 0.091 μM, 1.6 μM, 1.8 μM and 1.6 μM, respectively. In addition, the Kd of Nocodazole for Abl(E255K) phosphorylated, Abl(T315I) phosphorylated, BRAF(V600E) and PI3Kγ is 0.12 μM, 0.17 μM, 1.1 μM and 1.5 μM, respectively. Nocodazole induces apoptosis in chronic lymphocytic leukemia cells. Nocodazole inhibits insulin-stimulated glucose transport. Nocodazole decreases apoptosis in some human colon carcinoma cells. Nocodazole impairs the morphology and directionality of migrating medial gan-glionic eminence cells. [1] At high concentrations, Nocodazole rapidly depolymerizes microtubules in cells, while low concentrations of Nocodazole inhibit microtubule dynamic instability. [2] Mitotic cells incubated with different concentrations of Paclitaxel are inhibited from progressing to G1 phase 6 hours after release from the Nocodazole block, with a median inhibitory concentration of 4 nM. Nocodazole-pretreated cells exposed to Paclitaxel in the absence of Nocodazole only form free-floating microtubules, whereas pretreated cells exposed to Paclitaxel in the presence of Nocodazole-assembled centrosome organize microtubules. [3] Nocodazole disrupts microtubules by binding to β-tubulin. Nocodazole prevents the formation of one of the two interchain disulfide linkages. Nocodazole impairs the transport of vesicles. Nocodazole suppress METH-induced cell death and lysosomal dysfunction. METH-induced cell death is significantly decreased by Nocodazole pretreatment in comparison to METH alone. [4]Nocodazole doubles HDR efficiency to up to 30% in iPSCs. It improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing[6].

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
rat LT12 cells M4H5dnBzd2yrZnXyZZRqd25iYYPzZZk> MXW0PEBp MkSwRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDyZZQhVFRzMjDj[YxteyCjZoTldkA1QCCqcoOgZpkhYFSWIHHzd4F6NCCLQ{WwQVYhdk1? M1r4T|E6OjJyMEG4
human PANC1 cells NV7pU4I3WHKxbHnm[ZJifGmxbjDhd5NigQ>? NYHOdW1LPDhiaB?= MnniRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCSQV7DNUBk\WyuczDh[pRmeiB2ODDodpMh[nliU2LCJIF{e2G7LDDHTVUxRTFyIH7N MlLkNlU5Ojd3MkK=
human HeLa cells NGOyboNRem:uaX\ldoF1cW:wIHHzd4F6 MmPWOFghcA>? Mn\CRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCKZVzhJINmdGy|IHHmeIVzKDR6IHjyd{BjgSCVUlKgZZN{[XluIFfJOVA:OTVibl2= M4nteFI2QDJ5NUKy
human MDA-MB-231 cells NWPpelFEWHKxbHnm[ZJifGmxbjDhd5NigQ>? M4LyeFQ5KGh? NHzRNnZCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIF3ERU1OSi1{M{GgZ4VtdHNiYX\0[ZIhPDhiaILzJIJ6KFOUQjDhd5NigSxiR1m1NF0yPyCwTR?= MUCyOVgzPzV{Mh?=
human A549 cells NWPOW3hlWHKxbHnm[ZJifGmxbjDhd5NigQ>? NXrmZnYxPDhiaB?= NVjueIU2SW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDBOVQ6KGOnbHzzJIFnfGW{IES4JIhzeyCkeTDTVmIh[XO|YYmsJGdKPTB;MkCgcm0> MkXUNlU5Ojd3MkK=
lymphoid leukemia L1210 cells M2j3U3Bzd2yrZnXyZZRqd25iYYPzZZk> M{PBXVQ5KGh? M2LxR2lvcGmkaYTvdpkh[2:wY3XueJJifGmxbjDh[4FqdnO2IIDyc4xq\mW{YYTpc44hd2ZiY4XseJVz\WRibIntdIhwcWRibHX1b4VucWFiTEGyNVAh[2WubIOg[JVzcW6pIES4JIhzeyxiSVS1NF0zPyCwTR?= NV;jRZVrPzF|MUS4Ny=>
human A549 cells  Ml;6R5l1d3SxeHnjxsBie3OjeR?= NXi2RoI3OjRiaB?= NFLTcWtEgXSxdH;4bYNqfHliYXfhbY5{fCCqdX3hckBCPTR7IHPlcIx{KGGodHXyJFI1KGi{czDifUBOXFRiYYPzZZktKEmFNUC9N|YvPiCwTR?= NUn3WJhNOjBzMUCxN|c>
L1210 murine leukemia cell M3POdGdzd3e2aDDpcohq[mm2aX;uJIF{e2G7 M37pdmlvKH[rdILvJIZweiCrdIOgbY5pcWKrdH;yfUBi[3Srdnn0fUBi\2GrboP0JGwyOjFyIH31dolv\SCuZYXr[Y1q[SClZXzsJIdzd3e2aDygTWM2OD1|ODDuUS=> NH31Sm0yPzN6MUS2
mouse P388 cells NGTXRnpRem:uaX\ldoF1cW:wIHHzd4F6 M3Ht[lQ5KGh? M3jHdmFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgcY92e2ViUEO4PEBk\WyuczDh[pRmeiB2ODDodpMh[nliWGTUJIF{e2G7LDDJR|UxRTRyIH7N NYfGd3BLOTl{MkCwNVg>
LT12 cells NUTDZWtbWHKxbHnm[ZJifGmxbjDhd5NigQ>? NWixVGRzPDhiaB?= M1nMRmFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgUHQyOiClZXzsd{BjgSC[VGSgZZN{[XliYX\0[ZIhPDhiaILzMEBKSzVyPUSwJI5O NGHuZY8yPzF6MUG2OC=>
human U2OS cells MUDGeY5kfGmxbjDhd5NigQ>? MYe3NkBp M1TtPWlv\HWldHnvckBw\iCvaYTveIlkKGG{cnXzeEBqdiCqdX3hckBWOk:VIHPlcIx{KGG|c3Xzd4VlKGG|IH3peI91cWNiaX7k[Zgh[W[2ZYKgO|IhcHK|IHL5JI1q[3Kxc3PvdIlkKGGwYXz5d4l{NCCHQ{WwQVUxKG6P NIjQemkzPDlyMEi4Oy=>
human NCI-H460 cells NYj1NWNLWHKxbHnm[ZJifGmxbjDhd5NigQ>? M2X2[FQ5KGh? MVjBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKE6FST3IOFYxKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD14ODDuUS=> MUiyNVU3Ozd3MB?=
human BT549 cells NGfGOFhRem:uaX\ldoF1cW:wIHHzd4F6 NYHF[VlDPDhiaB?= NFztSoVCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIFLUOVQ6KGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD14OTDuUS=> NIrHepIzOTV4M{e1NC=>
human 451LU cells NUP1VGVJWHKxbHnm[ZJifGmxbjDhd5NigQ>? MXe0PEBp M2fFeWFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iNEWxUHUh[2WubIOgZYZ1\XJiNEigbJJ{KGK7IHHsZY1ieiCkbIXlJIF{e2G7LDDJR|UxRTd{IH7N NIrte2UzOTV4M{e1NC=>
human SKOV3 cells M4T1OXBzd2yrZnXyZZRqd25iYYPzZZk> NV3rbGxmPDhiaB?= MnSxRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCVS1;WN{Bk\WyuczDh[pRmeiB2ODDodpMh[nliYXzhcYFzKGKudXWgZZN{[XluIFnDOVA:QDBibl2= NVjBUFQyOjF3NkO3OVA>
K562 cells NVq3Noc1TnWwY4Tpc44h[XO|YYm= MYPF[oZm[3SrdnWgZ49v[2WwdILheIlwdiC{ZYH1bZJm\CC2bzDhdpJme3RicILvdIllcXWvIHnv[Ill\SC|dHHpcoVlKEt3NkKgZ4VtdHNiaX6gS|IwVSCyaHHz[UwhTUN3ME24NkBvVQ>? Ml3lNVI5PTJ5Nki=
human SKBR3 cells NGrxVo5Iem:5dHigbY5pcWKrdHnvckBie3OjeR?= MXS0PEBp NH7DUWRIem:5dHigbY5pcWKrdHnvckBw\iCqdX3hckBUU0KUMzDj[YxteyCjZoTldkA1QCCqcoOgZpkhVVSWIHHzd4F6 MVmyNlg2ODJzNB?=
human COLO205 cells MnHyVJJwdGmoZYLheIlwdiCjc4PhfS=> NYjzW2I3PDhiaB?= M{n4TGFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iQ1;MU|IxPSClZXzsd{Bi\nSncjC0PEBpenNiYomgZYxidWG{IHLseYUh[XO|YYmgMEBKSzVyPUi4JI5O M{jFWlIyPTZ|N{Ww
human KB/HeLa cells NFPkNnFHfW6ldHnvckBie3OjeR?= MWOyOEBp NYrMTolOS2WubDDjfYNt\SCjcoLld5QhcW5iaIXtZY4hU0JxSHXMZUBk\WyuczDhd5Nme3OnZDDhd{BIOi:PIIDoZZNmKGGlY4XteYxifGmxbjDh[pRmeiB{NDDodpMh[nliRlHDV{BidmGueYPpd{whTUN3ME25NUBvVQ>? MUSxPVIzODBzOB?=
human A549 cells MkTtS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? MYfHdo94fGhiaX7obYJqfGmxbjDv[kBpfW2jbjDBOVQ6KGOnbHzzJIJ6KFO3bH\vdohw\GGvaX7lJGIh[XO|YYmsJGdKPTB;MD6xJO69VQ>? M3r1dVIyOTB4NEW4
human MCF7 cells M4i3[2dzd3e2aDDpcohq[mm2aX;uJIF{e2G7 MUXHdo94fGhiaX7obYJqfGmxbjDv[kBpfW2jbjDNR2Y4KGOnbHzzJIJ6KFO3bH\vdohw\GGvaX7lJGIh[XO|YYmsJGdKPTB;MD6xJO69VQ>? NHjHcZQzOTFyNkS1PC=>
human RKO cells NHrreJlRem:uaX\ldoF1cW:wIHHzd4F6 NWHKfXVbSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDST28h[2WubIOgZpkhYFSWIHHzd4F6NCCLQ{WwQVAvOTFizszN M122[|E6OjJyMEG4
human SW480 cells MmTRVJJwdGmoZYLheIlwdiCjc4PhfS=> Mk\rOFghcA>? NGXHW4RCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIGPXOFgxKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD1yLkGyJO69VQ>? M2DKVFIyPTZ|N{Ww
human NCI-H460 cells NUDWUWNDWHKxbHnm[ZJifGmxbjDhd5NigQ>? NWSwV4RMPDhiaB?= NXzKfHlvSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDOR2kuUDR4MDDj[YxteyCjZoTldkA1QCCqcoOgZpkhYFSWIHHzd4F6NCCLQ{WwQVAvOTVizszN M1jLeFIxPTN5N{[1
human SF268 cells NVPNU5FzS3m2b4TvfIlkyqCjc4PhfS=> NHXIPYI1QCCq NIfD[IREgXSxdH;4bYNqfHliYXfhbY5{fCCqdX3hckBUTjJ4ODDj[YxteyCjZoTldkA1QCCqcoOgZpkhYFSWIHHzd4F6NCCLQ{WwQVAvOyEQvF2= NX\nUlVsOjF5MEWyNlM>
human SK-N-SH cells Mnj3S5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? MXrHdo94fGhiaX7obYJqfGmxbjDv[kBpfW2jbjDTT{1PNVOKIHPlcIx{KGK7IGP1cIZwemixZHHtbY5mKEJiYYPzZZktKEeLNUC9NE43PiEQvF2= MVuyNVExPjR3OB?=
human MIAPaCa2 cells Mnr2VJJwdGmoZYLheIlwdiCjc4PhfS=> MkfXOFghcA>? MlLRRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCPSVHQZWNiOiClZXzsd{Bie3Onc4Pl[EBieyCpcn;3eIghcW6qaXLpeIlwdiCjZoTldkA1QCCqcoOgZpkhW1KEIHHzd4F6NCCJSUWwQVAvQTVizszN MkC0NlUyOzF7NU[=
human HaCaT cells MXnDfZRwfG:6aXRCpIF{e2G7 MkTsOFghcA>? NY\rbZVjS3m2b4TvfIlkKGGldHn2bZR6KGGpYXnud5QhcHWvYX6gTIFE[VRiY3XscJMh[XO|ZYPz[YQh[XNicnXkeYN1cW:wIHnuJINmdGxiZ4Lve5RpKGmwY4XiZZRm\CCob4KgOFghcHK|IHL5JG1VXCCjc4PhfUwhUUN3ME24Mlkh|ryP MW[yOlI{OTB6MB?=
human SGC7901 cells NHHXSXNHfW6ldHnvckBie3OjeR?= MYm0PEBp NFnrSJNKdmS3Y4Tpc44hd2ZiZHnzeI9zfGmxboOgbY4hfHWkdXzpckBie3OnbXLsfUBqdiCqdX3hckBUT0N5OUCxJINmdGy|IHHzd4V{e2WmIHHzJIFjdm:{bXHsJI1qfG:2aXOgd5BqdmSuZTDmc5Ju[XSrb36gZZQhOiCob3zkJGlEPTBibHX2[YwhcW6ldXLheIVlKG[xcjC0PEBpenNiYomgbY1ufW6xZnz1c5Jme2OnbnPlJJN1[WmwaX7nJIJie2WmIH\seY9z\XOlZX7j[UBucWO{b4Pjc5B6 MYGyOVI3OjB3MR?=
human Jurkat T cells MYLGeY5kfGmxbjDhd5NigQ>? NUCyV|VYOzZiaB?= NHm4[2VKdmS3Y4Tpc44hd2ZibXnjdo91fWK3bHWg[IFu[WenIHnuJIh2dWGwIFr1dotifCCWIHPlcIx{KG:4ZYLlfJBz\XO|aX7nJGJkdDJiYX\0[ZIhOzZiaILzJIJ6KGmvbYXuc5N1[WmwaX7nJI1mfGixZB?= NGTMR3EzOjF7N{O5Ny=>
human PC3M cells NXi2T2ZCTnWwY4Tpc44h[XO|YYm= NYG4dHJ3OC53IIXnM41t M2PQcVI1KGh? NGnRd4VKdmS3Y4Tpc44hd2ZiYYDvdJRwe2m|IHnuJIh2dWGwIGDDN20h[2WubIOgZZN{\XO|ZXSgZZMh[WOldX31cIF1cW:wIHH0JJN2[kdzIIDoZZNmKGG2IECuOUB2\y:vbDDh[pRmeiB{NDDodpMh[nliZnzve{BkgXSxbXX0dpk> M33GbVE5OjR5NUez
mouse NIH3T3 cells NG[yPXNHfW6ldHnvckBie3OjeR?= NGTwTJcxNjVidXevcYw> M3\Ib|I1KGh? MVjJcoR2[3Srb36gc4Yh[XCxcITvd4l{KGmwIH3veZNmKE6LSEPUN{Bk\WyuczDhd5Nme3OnZDDhd{Bi[2O3bYXsZZRqd25iYYSgd5VjTzFicHjhd4Uh[XRiMD61JJVoN22uIHHmeIVzKDJ2IHjyd{BjgSCobH;3JIN6fG:vZYTyfS=> NVrheIFVOTh{NEe1O|M>

... Click to View More Cell Line Experimental Data

Assay
Methods Test Index PMID
Western blot
Cdc2 / cyclin B1 / MAD2 / Cdc20 ; 

PubMed: 21918689     


E (upper part). Time-dependent changes in cyclin B1 and Cdc2 protein levels following nocodazole treatment. Cells were treated with nocodazole (250 nM) for the length of time as indicated, and whole cell lysates were prepared. An equal amount of protein lysates was electrophoretically separated on the 10% SDS-polyacrylamide gel, and transferred to nitrocellulose membrane. Western blots were detected using specific antibodies against cyclin B1, Cdc2 (CDK1), MAD2, and Cdc20 on an enhanced chemiluminescence (ECL) apparatus. Membrane was stripped for determining the levels of GAPDH as a loading control. E (lower part). The relative protein levels for cyclin B1, Cdc2, MAD2, and Cdc20 were calculated according to their densitometry readings, which were normalized according to the corresponding readings for the GAPDH protein bands. Each value is mean ± S.D. from three replicate determinations. * P<0.05, ** P<0.01 versus vehicle-treated control. 

21918689
Immunofluorescence
Tubulin / LC8 ; 

PubMed: 23038268     


Shown is endogenous LC8 colocalized with microtubules in HeLa cells. Control, 20 nm taxol- and 100 nm nocodazole-treated cells were stained with α-tubulin and LC8 antibodies. Scale bar, 20 μm.

Brd4 / α-tubulin ; 

PubMed: 22567088     


Cells were treated with indicated concentrations of nocodazole for 4 h, and mitotic cells were immunostained for endogenous Brd4 and α-tubulin. DNA was stained by Hoechst33324.

23038268 22567088
Growth inhibition assay
Cell viability ; 

PubMed: 21918689     


Changes in cell viability (MTT assay) after treatment of cells with 250 nM nocodazole for different lengths of time. Each data point is the mean ± S.D. from four replicate measurements from one representative experiment. 

21918689
In vivo The tumor volume and tumor weight of the mice treated with Ketoconazole  plus  Nocodazole are significantly reduced as compared with those treated with Ketoconazole or Nocodazole alone. Combined treatment with Ketoconazole  plus  Nocodazole strongly enhances apoptosis of COLO 205 tumor xenografts treated with Ketoconazole  or  Nocodazole alone. [5]

Protocol

Animal Research:

[5]

- Collapse
  • Animal Models: Nude mice with COLO-205 tumor xenografts
  • Formulation: 0.05% dimethyls
  • Dosages: 5 mg/kg
  • Administration: Administered via i.p.
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 7 mg/mL (23.23 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents to the product individually and in order(Data is from Selleck tests instead of citations):
2% DMSO+30% PEG 300+2% Tween 80+ddH2O
For best results, use promptly after mixing.
1mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 301.32
Formula

C14H11N3O3S

CAS No. 31430-18-9
Storage powder
in solvent
Synonyms Oncodazole

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Microtubule Associated Signaling Pathway Map

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID