Nocodazole

Catalog No.S2775 Synonyms: Oncodazole

Nocodazole Chemical Structure

Molecular Weight(MW): 301.32

Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.

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Cited by 22 Publications

Purity & Quality Control

Choose Selective Microtubule Associated Inhibitors

Biological Activity

Description Nocodazole is a rapidly-reversible inhibitor of microtubule polymerization, also inhibits Abl, Abl(E255K) and Abl(T315I) with IC50 of 0.21 μM, 0.53 μM and 0.64 μM in cell-free assays, respectively. Nocodazole improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing.
Targets
Microtubules [2]
(Cell-free assay)
Abl [1]
(Cell-free assay)
Abl (E255K) [1]
(Cell-free assay)
Abl (T315I) [1]
(Cell-free assay)
0.21 μM 0.53 μM 0.64 μM
In vitro

Nocodazole is a high-affinity ligand for the cancer-related kinases including Abl phosphorylated, c-Kit, BRAF, and MEK with Kd of 0.091 μM, 1.6 μM, 1.8 μM and 1.6 μM, respectively. In addition, the Kd of Nocodazole for Abl(E255K) phosphorylated, Abl(T315I) phosphorylated, BRAF(V600E) and PI3Kγ is 0.12 μM, 0.17 μM, 1.1 μM and 1.5 μM, respectively. Nocodazole induces apoptosis in chronic lymphocytic leukemia cells. Nocodazole inhibits insulin-stimulated glucose transport. Nocodazole decreases apoptosis in some human colon carcinoma cells. Nocodazole impairs the morphology and directionality of migrating medial gan-glionic eminence cells. [1] At high concentrations, Nocodazole rapidly depolymerizes microtubules in cells, while low concentrations of Nocodazole inhibit microtubule dynamic instability. [2] Mitotic cells incubated with different concentrations of Paclitaxel are inhibited from progressing to G1 phase 6 hours after release from the Nocodazole block, with a median inhibitory concentration of 4 nM. Nocodazole-pretreated cells exposed to Paclitaxel in the absence of Nocodazole only form free-floating microtubules, whereas pretreated cells exposed to Paclitaxel in the presence of Nocodazole-assembled centrosome organize microtubules. [3] Nocodazole disrupts microtubules by binding to β-tubulin. Nocodazole prevents the formation of one of the two interchain disulfide linkages. Nocodazole impairs the transport of vesicles. Nocodazole suppress METH-induced cell death and lysosomal dysfunction. METH-induced cell death is significantly decreased by Nocodazole pretreatment in comparison to METH alone. [4]Nocodazole doubles HDR efficiency to up to 30% in iPSCs. It improves the CRISPR-mediated HDR efficiency and has an additive effect on enhancing precise genome editing[6].

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
rat LT12 cells MmSwVJJwdGmoZYLheIlwdiCjc4PhfS=> M1vHOVQ5KGh? MX;BcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IILheEBNXDF{IHPlcIx{KGGodHXyJFQ5KGi{czDifUBZXFRiYYPzZZktKEmFNUC9OkBvVQ>? M1HrWlE6OjJyMEG4
human PANC1 cells NEXVNZdRem:uaX\ldoF1cW:wIHHzd4F6 MlzuOFghcA>? NUT5emxPSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDQRW5EOSClZXzsd{Bi\nSncjC0PEBpenNiYomgV3JDKGG|c3H5MEBIUTVyPUGwJI5O NHvpO3YzPTh{N{WyNi=>
human HeLa cells MXnQdo9tcW[ncnH0bY9vKGG|c3H5 MUO0PEBp NX7VVIZ7SW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDI[WxiKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDTVmIh[XO|YYmsJGdKPTB;MUWgcm0> NGTrTYEzPTh{N{WyNi=>
human MDA-MB-231 cells NFnwZ4hRem:uaX\ldoF1cW:wIHHzd4F6 M2nSTFQ5KGh? NVLyWXdbSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDNSGEuVUJvMkOxJINmdGy|IHHmeIVzKDR6IHjyd{BjgSCVUlKgZZN{[XluIFfJOVA:OTdibl2= NHrTVY4zPTh{N{WyNi=>
human A549 cells MXXQdo9tcW[ncnH0bY9vKGG|c3H5 NES2cVA1QCCq M3z0RmFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iQUW0PUBk\WyuczDh[pRmeiB2ODDodpMh[nliU2LCJIF{e2G7LDDHTVUxRTJyIH7N M4WxV|I2QDJ5NUKy
lymphoid leukemia L1210 cells NWf6Nnl4WHKxbHnm[ZJifGmxbjDhd5NigQ>? M2DjeFQ5KGh? NXj0cndCUW6qaXLpeI9zgSClb37j[Y51emG2aX;uJIFo[Wmwc4SgdJJwdGmoZYLheIlwdiCxZjDjeYx1fXKnZDDsfY1xcG:rZDDs[ZVs\W2rYTDMNVIyOCClZXzsd{BlfXKrbnegOFghcHK|LDDJSFUxRTJ5IH7N MUi3NVMyPDh|
human A549 cells  NXfEV5VUS3m2b4TvfIlkyqCjc4PhfS=> Mm\tNlQhcA>? MXnDfZRwfG:6aXPpeJkh[WejaX7zeEBpfW2jbjDBOVQ6KGOnbHzzJIFnfGW{IEK0JIhzeyCkeTDNWHQh[XO|YYmsJGlEPTB;M{[uOkBvVQ>? MVOyNFEyODF|Nx?=
L1210 murine leukemia cell MnnyS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NWfZN4FXUW5idnn0do8h\m:{IHn0d{BqdmirYnn0c5J6KGGldHn2bZR6KGGpYXnud5QhVDF{MUCgcZVzcW6nIHzleYtmdWmjIHPlcIwh\3Kxd4ToMEBKSzVyPUO4JI5O MniyNVc{QDF2Nh?=
mouse P388 cells NXPMVGNCWHKxbHnm[ZJifGmxbjDhd5NigQ>? MknxOFghcA>? MoqxRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDtc5V{\SCSM{i4JINmdGy|IHHmeIVzKDR6IHjyd{BjgSC[VGSgZZN{[XluIFnDOVA:PDBibl2= MW[xPVIzODBzOB?=
LT12 cells NFrERlJRem:uaX\ldoF1cW:wIHHzd4F6 NYrKUIxmPDhiaB?= NWLpdndMSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBNXDF{IHPlcIx{KGK7IGjUWEBie3OjeTDh[pRmeiB2ODDodpMtKEmFNUC9OFAhdk1? NIPGZW0yPzF6MUG2OC=>
human U2OS cells MoLlSpVv[3Srb36gZZN{[Xl? NWLIOJk6PzJiaB?= M2\kUWlv\HWldHnvckBw\iCvaYTveIlkKGG{cnXzeEBqdiCqdX3hckBWOk:VIHPlcIx{KGG|c3Xzd4VlKGG|IH3peI91cWNiaX7k[Zgh[W[2ZYKgO|IhcHK|IHL5JI1q[3Kxc3PvdIlkKGGwYXz5d4l{NCCHQ{WwQVUxKG6P MojBNlQ6ODB6OEe=
human NCI-H460 cells MkDtVJJwdGmoZYLheIlwdiCjc4PhfS=> NFr0fY81QCCq NF6xc2VCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIF7DTU1JPDZyIHPlcIx{KGGodHXyJFQ5KGi{czDifUBidGGvYYKgZox2\SCjc4PhfUwhUUN3ME22PEBvVQ>? NWjqNFVWOjF3NkO3OVA>
human BT549 cells NYTWTIdYWHKxbHnm[ZJifGmxbjDhd5NigQ>? NYDJemQ3PDhiaB?= MmDsRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDoeY1idiCEVEW0PUBk\WyuczDh[pRmeiB2ODDodpMh[nliYXzhcYFzKGKudXWgZZN{[XluIFnDOVA:Pjlibl2= MlW4NlE2PjN5NUC=
human 451LU cells MkDBVJJwdGmoZYLheIlwdiCjc4PhfS=> NFjXT5o1QCCq M1;tPGFvfGmycn;sbYZmemG2aY\lJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iNEWxUHUh[2WubIOgZYZ1\XJiNEigbJJ{KGK7IHHsZY1ieiCkbIXlJIF{e2G7LDDJR|UxRTd{IH7N NWPkWmVNOjF3NkO3OVA>
human SKOV3 cells MnLVVJJwdGmoZYLheIlwdiCjc4PhfS=> M1HRNVQ5KGh? NGq0SmtCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIGPLU3Y{KGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD16MDDuUS=> NGTLRVczOTV4M{e1NC=>
K562 cells M1W3XGZ2dmO2aX;uJIF{e2G7 M4PVNGVn\mWldHn2[UBkd26lZX70doF1cW:wIILldZVqemWmIITvJIFzemW|dDDwdo9xcWSrdX2gbY9lcWSnIIP0ZYlv\WRiS{W2NkBk\WyuczDpckBIOi:PIIDoZZNmNCCHQ{WwQVgzKG6P NFS3N|IyOjh3Mke2PC=>
human SKBR3 cells NHrBSINIem:5dHigbY5pcWKrdHnvckBie3OjeR?= NH;veY01QCCq NIewWHpIem:5dHigbY5pcWKrdHnvckBw\iCqdX3hckBUU0KUMzDj[YxteyCjZoTldkA1QCCqcoOgZpkhVVSWIHHzd4F6 NXfQU|R7OjJ6NUCyNVQ>
human COLO205 cells NGfXW3BRem:uaX\ldoF1cW:wIHHzd4F6 Mlv6OFghcA>? MULBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKEORTF:yNFUh[2WubIOgZYZ1\XJiNEigbJJ{KGK7IHHsZY1ieiCkbIXlJIF{e2G7IDygTWM2OD16ODDuUS=> NVHEcZdiOjF3NkO3OVA>
human KB/HeLa cells NGn4RWdHfW6ldHnvckBie3OjeR?= NVe2O4oxOjRiaB?= NUP0VmF6S2WubDDjfYNt\SCjcoLld5QhcW5iaIXtZY4hU0JxSHXMZUBk\WyuczDhd5Nme3OnZDDhd{BIOi:PIIDoZZNmKGGlY4XteYxifGmxbjDh[pRmeiB{NDDodpMh[nliRlHDV{BidmGueYPpd{whTUN3ME25NUBvVQ>? M{HjflE6OjJyMEG4
human A549 cells MV\Hdo94fGhiaX7obYJqfGmxbjDhd5NigQ>? Mn3xS5Jwf3SqIHnubIljcXSrb36gc4YhcHWvYX6gRVU1QSClZXzsd{BjgSCVdXzmc5Jpd2SjbXnu[UBDKGG|c3H5MEBIUTVyPUCuNUDPxE1? MYGyNVExPjR3OB?=
human MCF7 cells Ml2yS5Jwf3SqIHnubIljcXSrb36gZZN{[Xl? NYXsSXE6T3Kxd4ToJIlvcGmkaYTpc44hd2ZiaIXtZY4hVUOINzDj[YxteyCkeTDTeYxnd3Kqb3ThcYlv\SCEIHHzd4F6NCCJSUWwQVAvOSEQvF2= MV[yNVExPjR3OB?=
human RKO cells NWnEO4NKWHKxbHnm[ZJifGmxbjDhd5NigQ>? NFXLR2tCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIGLLU{Bk\WyuczDifUBZXFRiYYPzZZktKEmFNUC9NE4yOSEQvF2= MYCxPVIzODBzOB?=
human SW480 cells NYDmRm9yWHKxbHnm[ZJifGmxbjDhd5NigQ>? M3;uUlQ5KGh? NFvZOXBCdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JIh2dWGwIGPXOFgxKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDhcIFu[XJiYnz1[UBie3OjeTygTWM2OD1yLkGyJO69VQ>? MojINlE2PjN5NUC=
human NCI-H460 cells MV\Qdo9tcW[ncnH0bY9vKGG|c3H5 NFX3U4k1QCCq MXTBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IHj1cYFvKE6FST3IOFYxKGOnbHzzJIFnfGW{IES4JIhzeyCkeTDYWHQh[XO|YYmsJGlEPTB;MD6xOUDPxE1? M2Kz[lIxPTN5N{[1
human SF268 cells NXzZNnk5S3m2b4TvfIlkyqCjc4PhfS=> M3fyV|Q5KGh? NVL5V45QS3m2b4TvfIlkcXS7IHHnZYlve3RiaIXtZY4hW0Z{NkigZ4VtdHNiYX\0[ZIhPDhiaILzJIJ6KFiWVDDhd5NigSxiSVO1NF0xNjNizszN MWqyNVcxPTJ{Mx?=
human SK-N-SH cells NFHmNVRIem:5dHigbY5pcWKrdHnvckBie3OjeR?= MX\Hdo94fGhiaX7obYJqfGmxbjDv[kBpfW2jbjDTT{1PNVOKIHPlcIx{KGK7IGP1cIZwemixZHHtbY5mKEJiYYPzZZktKEeLNUC9NE43PiEQvF2= NXi3eIROOjFzME[0OVg>
human MIAPaCa2 cells NFe2ZllRem:uaX\ldoF1cW:wIHHzd4F6 NF7KeGM1QCCq NXfjOVJ6SW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBpfW2jbjDNTWFR[UOjMjDj[YxteyCjc4Pld5Nm\CCjczDndo94fGhiaX7obYJqfGmxbjDh[pRmeiB2ODDodpMh[nliU2LCJIF{e2G7LDDHTVUxRTBwOUWg{txO NYHmZWViOjVzM{G5OVY>
human HaCaT cells NHLxb5BEgXSxdH;4bYPDqGG|c3H5 MljkOFghcA>? MojPR5l1d3SxeHnjJIFkfGm4aYT5JIFo[Wmwc4SgbJVu[W5iSHHDZXQh[2WubIOgZZN{\XO|ZXSgZZMhemWmdXP0bY9vKGmwIHPlcIwh\3Kxd4ToJIlv[3WkYYTl[EBnd3JiNEigbJJ{KGK7IF3UWEBie3OjeTygTWM2OD16Lkmg{txO NX3CUllWOjZ{M{GwPFA>
human SGC7901 cells NVzH[nVGTnWwY4Tpc44h[XO|YYm= M3\NNFQ5KGh? MVTJcoR2[3Srb36gc4Yh\Gm|dH;yeIlwdnNiaX6geJVjfWyrbjDhd5NmdWKueTDpckBpfW2jbjDTS2M4QTBzIHPlcIx{KGG|c3Xzd4VlKGG|IHHico9zdWGuIH3peI91cWNic4DpcoRt\SCob4LtZZRqd25iYYSgNkBnd2ymIFnDOVAhdGW4ZXygbY5kfWKjdHXkJIZweiB2ODDodpMh[nliaX3teY5w\my3b4Lld4NmdmOnIIP0ZYlvcW6pIHLhd4VlKG[udX;y[ZNk\W6lZTDtbYNzd3Olb4D5 NGrqVpEzPTJ4MkC1NS=>
human Jurkat T cells NU\a[2FCTnWwY4Tpc44h[XO|YYm= NYXqV|NSOzZiaB?= NGnK[mFKdmS3Y4Tpc44hd2ZibXnjdo91fWK3bHWg[IFu[WenIHnuJIh2dWGwIFr1dotifCCWIHPlcIx{KG:4ZYLlfJBz\XO|aX7nJGJkdDJiYX\0[ZIhOzZiaILzJIJ6KGmvbYXuc5N1[WmwaX7nJI1mfGixZB?= NF30Z2IzOjF7N{O5Ny=>
human PC3M cells Ml;zSpVv[3Srb36gZZN{[Xl? MXmwMlUhfWdxbXy= MXeyOEBp MYHJcoR2[3Srb36gc4Yh[XCxcITvd4l{KGmwIHj1cYFvKFCFM12gZ4VtdHNiYYPz[ZN{\WRiYYOgZYNkfW23bHH0bY9vKGG2IIP1ZmcyKHCqYYPlJIF1KDBwNTD1[{9udCCjZoTldkAzPCCqcoOgZpkh\myxdzDjfZRwdWW2com= M{LQelE5OjR5NUez
mouse NIH3T3 cells MX;GeY5kfGmxbjDhd5NigQ>? MUewMlUhfWdxbXy= NULsTnBZOjRiaB?= NGnOTYVKdmS3Y4Tpc44hd2ZiYYDvdJRwe2m|IHnuJI1wfXOnIF7JTFNVOyClZXzsd{Bie3Onc4Pl[EBieyCjY3P1cZVt[XSrb36gZZQhe3WkR{GgdIhie2ViYYSgNE42KHWpL33sJIFnfGW{IEK0JIhzeyCkeTDmcI94KGO7dH;t[ZRzgQ>? M2HM[FE5OjR5NUez

... Click to View More Cell Line Experimental Data

Assay
Methods Test Index PMID
Western blot
Cdc2 / cyclin B1 / MAD2 / Cdc20 ; 

PubMed: 21918689     


E (upper part). Time-dependent changes in cyclin B1 and Cdc2 protein levels following nocodazole treatment. Cells were treated with nocodazole (250 nM) for the length of time as indicated, and whole cell lysates were prepared. An equal amount of protein lysates was electrophoretically separated on the 10% SDS-polyacrylamide gel, and transferred to nitrocellulose membrane. Western blots were detected using specific antibodies against cyclin B1, Cdc2 (CDK1), MAD2, and Cdc20 on an enhanced chemiluminescence (ECL) apparatus. Membrane was stripped for determining the levels of GAPDH as a loading control. E (lower part). The relative protein levels for cyclin B1, Cdc2, MAD2, and Cdc20 were calculated according to their densitometry readings, which were normalized according to the corresponding readings for the GAPDH protein bands. Each value is mean ± S.D. from three replicate determinations. * P<0.05, ** P<0.01 versus vehicle-treated control. 

21918689
Immunofluorescence
Tubulin / LC8 ; 

PubMed: 23038268     


Shown is endogenous LC8 colocalized with microtubules in HeLa cells. Control, 20 nm taxol- and 100 nm nocodazole-treated cells were stained with α-tubulin and LC8 antibodies. Scale bar, 20 μm.

Brd4 / α-tubulin ; 

PubMed: 22567088     


Cells were treated with indicated concentrations of nocodazole for 4 h, and mitotic cells were immunostained for endogenous Brd4 and α-tubulin. DNA was stained by Hoechst33324.

23038268 22567088
Growth inhibition assay
Cell viability ; 

PubMed: 21918689     


Changes in cell viability (MTT assay) after treatment of cells with 250 nM nocodazole for different lengths of time. Each data point is the mean ± S.D. from four replicate measurements from one representative experiment. 

21918689
In vivo The tumor volume and tumor weight of the mice treated with Ketoconazole  plus  Nocodazole are significantly reduced as compared with those treated with Ketoconazole or Nocodazole alone. Combined treatment with Ketoconazole  plus  Nocodazole strongly enhances apoptosis of COLO 205 tumor xenografts treated with Ketoconazole  or  Nocodazole alone. [5]

Protocol

Animal Research:

[5]

- Collapse
  • Animal Models: Nude mice with COLO-205 tumor xenografts
  • Formulation: 0.05% dimethyls
  • Dosages: 5 mg/kg
  • Administration: Administered via i.p.
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 7 mg/mL (23.23 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents to the product individually and in order(Data is from Selleck tests instead of citations):
2% DMSO+30% PEG 300+2% Tween 80+ddH2O
For best results, use promptly after mixing.
1mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 301.32
Formula

C14H11N3O3S

CAS No. 31430-18-9
Storage powder
in solvent
Synonyms Oncodazole

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Microtubule Associated Signaling Pathway Map

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID