Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

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Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
C41 MmHpT4lv[XOnIFHzd4F6 M1f1[|MxKG2rbh?= NVHPPYltUW6qaXLpeIlwdiCxZjDQRXJROSC5aYToJGVEPTBib3[gNE4xODJizszN M2rifVE6QDh6N{[w
Jurkat MkexT4lv[XOnIFHzd4F6 MkHmPVYhcA>? NUjiZm92TE2VTx?= MUHJcohq[mm2aX;uJI9nKFCDUmCxJIF{e2W|c3XkJIF{KHKnZIXjeIlwdiCxZjDj[YxtKH[rYXLpcIl1gSC5aYToJGVEPTBib3[gN{DPxE1? MW[yN|g2ODF7OR?=
Capan1 NYDUTYJIT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NW\mco1lPzJiaB?= MoTtSG1UVw>? MX7BcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IFLSR2EzKGenbnWgcZV1[XSnZDDoeY1idiCFYYDhclEh[2WubIOge4l1cCCLQ{WwJI9nKDN7Lkeg{txO MmH5NlQ{QTh|OEO=
DT40 M3v4SmN6fG:2b4jpZ{BCe3OjeR?= NYLNd25iPzJiaB?= NGHwOohFVVOR NIOwTpBEgXSxdH;4bYNqfHliYXfhbY5{fCClaHnjb4VvKEKUQ1GyMYRm\mmlaXXueEBFXDRyIHPlcIx{ M2TnSlI1QTJ{NUi3
ML-1 MUfBdI9xfG:2aXOgRZN{[Xl? NYq1RlhjOi53IN88US=> M4mzc|I1KGh? NVjJeYpMTE2VTx?= MoLDV5lv\XKpaYP0bYNidGy7IHXubIFv[2W|IGTSRWlNNWmwZIXj[YQh[XCxcITvd4l{KGmwIF3MMVEh[2WubIO= MmLONlQ5QTVzM{W=
HCT-116 MYrLbY5ie2ViQYPzZZk> NEO3TJoxNjVizszN MnHMNlQhcA>? MonoVGFTWCCjY4Tpeol1gSCmZXPy[YF{\XN? NEPoTHIzOzB3NEKxNy=>
UM-SCC1 MljCR5l1d3SxeHnjJGF{e2G7 M1fmZlExKM7:TR?= MXiyOEBp MWTS[YR2[2W|IITo[UBk\WyuII\pZYJqdGm2eR?= M3vuV|IyQTF{NkKw
FaDu NFHSdW1EgXSxdH;4bYMhSXO|YYm= NYLufodjOTBizszN M2LPcVI1KGh? MVfS[YR2[2W|IITo[UBk\WyuII\pZYJqdGm2eR?= NUCxN2gyOjF7MUK2NlA>
PC-3 Mnm4S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVyxNEDPxE1? MWfJcoR2[2W|IHGgd4lodmmoaXPhcpQhcW6qaXLpeIlwdiCrbjDjc4xwdnliZn;ycYF1cW:wwrC= MnrqNlE2PzF7MUK=
EoL-1-cell NGGzfVNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NVTVRYxHUUN3ME2xMlA4QThizszN M3\aV3NCVkeHUh?=
NCI-SNU-5 M{jMcmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWLJR|UxRTNwMUK4OFEh|ryP M4q5VnNCVkeHUh?=
BV-173 NYPpWJVxT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MnzoTWM2OD13LkS1OFA6KM7:TR?= Mn;NV2FPT0WU
HCC1806 M2qwWGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYHJR|UxRTVwN{WxO|Mh|ryP M373T3NCVkeHUh?=
COLO-680 MY\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NF[4UmZKSzVyPU[uNlE1ODZizszN MXHTRW5ITVJ?
NCI-H720 MmiwS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MX3JR|UxRThwNEO2NFMh|ryP MlvFV2FPT0WU
SK-MEL-1 MVnHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NEHiTlJKSzVyPUGyMlQ3PjNizszN M3m2d3NCVkeHUh?=
Ramos-2G6-4C10 M1S5NGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NI[yN|FKSzVyPUGyMlQ4PTJizszN M1fJUHNCVkeHUh?=
KY821 MmC2S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NIXoOYdKSzVyPUGyMlQ5PSEQvF2= MnfZV2FPT0WU
HEC-1 M1mxe2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWnJR|UxRTF{LkmxPVYh|ryP NXfPcnh2W0GQR1XS
MN-60 MXvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4L3PGlEPTB;MUOuOVM5QSEQvF2= M1TOc3NCVkeHUh?=
DU-145 NEK2OVhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NGXKUmtKSzVyPUGzMlkxPTNizszN M37PVHNCVkeHUh?=
EW-3 MnvSS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NFziUlNKSzVyPUG0MlU2PjVizszN NVfMNHhkW0GQR1XS
OS-RC-2 M1mzOGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MkLlTWM2OD1zNT65OVg6KM7:TR?= MkPaV2FPT0WU
RPMI-8226 MlGzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MXPJR|UxRTF4LkKwOFIh|ryP NXOxcYM3W0GQR1XS
ChaGo-K-1 M2DqdWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWHJR|UxRTF4LkWzNlUh|ryP MXzTRW5ITVJ?
DEL MYDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4HQdmlEPTB;MU[uOlcyPyEQvF2= M2n1V3NCVkeHUh?=
GP5d MknDS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnLOTWM2OD1zNz6wOVMh|ryP MXrTRW5ITVJ?
COLO-668 NXv4cXh1T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NULxNWRuUUN3ME2xO{43Ojl2IN88US=> Mo\5V2FPT0WU
H9 NWHmOJg2T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NIriPVlKSzVyPUG4MlI5OzNizszN M{DSWXNCVkeHUh?=
KYSE-150 NFvmcphIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M1vMWWlEPTB;MUiuPVk5PiEQvF2= MlXlV2FPT0WU
Daoy NEP0fWZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NGDnSnhKSzVyPUG5MlU3PDlizszN NWXSeXRZW0GQR1XS
ECC10 NGru[2ZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M2S0TmlEPTB;MkCuO|Q2PSEQvF2= MlrOV2FPT0WU
MHH-NB-11 MUfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M{jNS2lEPTB;MkOuNVM3OyEQvF2= MXnTRW5ITVJ?
HCC1937 MXrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NVvBUXNvUUN3ME2yOE44PDZizszN MmHqV2FPT0WU
TGBC11TKB MWrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4nZTGlEPTB;MkWuOlg3OyEQvF2= NF64WGdUSU6JRWK=
CTV-1 NXLDfVBwT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M1vMOmlEPTB;MkWuPFk3QSEQvF2= M3jIWnNCVkeHUh?=
NCI-H2029 NY\WR2p6T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NIDzfnhKSzVyPUK2MlQzOzhizszN NUfpRplNW0GQR1XS
NCI-H1693 MYfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MY\JR|UxRTJ5LkK4PVgh|ryP MXPTRW5ITVJ?
HCC70 MlPIS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NF36TmZKSzVyPUK3MlczPDZizszN MmG3V2FPT0WU
BEN MX\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NE\0S4FKSzVyPUK3Mlk2PjZizszN M4jZNnNCVkeHUh?=
LB771 NH\jdnNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M2\SOGlEPTB;MkiuPFM4OyEQvF2= MoHEV2FPT0WU
697 MneyS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NI\qeVZKSzVyPUK5MlAzOzVizszN NWHYeZJ2W0GQR1XS
LU-139 M4Sy[Gdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NYTsOXZjUUN3ME2yPU4{PzR6IN88US=> MYjTRW5ITVJ?
EW-13 MWfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFvsc4tKSzVyPUK5MlM5OTRizszN MWTTRW5ITVJ?
L-363 MUHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4fsTGlEPTB;MkmuOFc6QCEQvF2= MV3TRW5ITVJ?
EM-2 NETp[WlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NV\tU3Z3UUN3ME2yPU41QTBzIN88US=> NEDlVo1USU6JRWK=
RS4-11 M1riWWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M4HrZmlEPTB;M{CuOFI1OSEQvF2= M1rX[3NCVkeHUh?=
A2780 M{SyR2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M2rHRWlEPTB;M{CuO|Q2PyEQvF2= MVnTRW5ITVJ?
KU812 MlfjS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4WzV2lEPTB;M{KuN|Y1OiEQvF2= MWfTRW5ITVJ?
MFE-280 Mkn2S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NUXYS2lWUUN3ME2zN{4{QDh7IN88US=> MXLTRW5ITVJ?
JVM-3 M4PlS2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M2LTdGlEPTB;M{WuOVg3QCEQvF2= MlS0V2FPT0WU
MV-4-11 NXvNZodET3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NGLTN|dKSzVyPUO1Mlg1QTlizszN MWDTRW5ITVJ?
H4 MljOS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M{\zb2lEPTB;M{euOVY4KM7:TR?= MofBV2FPT0WU
T47D NI\4[ZJIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MXTJR|UxRTN5LkewNVgh|ryP NGPkSIVUSU6JRWK=
SW982 NXn5N4JPT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NUi4OnpQUUN3ME2zPE4xQTl6IN88US=> M2rnTnNCVkeHUh?=
IGROV-1 MonoS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVHJR|UxRTN7LkOzNFQh|ryP M{XnR3NCVkeHUh?=
NB14 NUXIOol[T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MWnJR|UxRTRyLkewN|Eh|ryP M13MOnNCVkeHUh?=
HCC1187 MlrGS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MUfJR|UxRTRzLkK3O|Eh|ryP MnH5V2FPT0WU
KARPAS-45 NVj1ZW9iT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MV3JR|UxRTRzLkS4NVgh|ryP NGLjd|lUSU6JRWK=
MOLT-4 M1nHdWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NH3vcplKSzVyPUSyMlI2OzhizszN NI\CVYVUSU6JRWK=
JVM-2 MXvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NF7obYRKSzVyPUSyMlkzODdizszN M4XhT3NCVkeHUh?=
A4-Fuk NFf4PJVIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NWTqPVFkUUN3ME20N{42PjlzIN88US=> M1:2[HNCVkeHUh?=
T98G MkL3S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NX3ST2JJUUN3ME20OE45PTF5IN88US=> M3XucnNCVkeHUh?=
Mo-T M37wfGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M2r6cGlEPTB;NEWuOlM5QSEQvF2= MmTRV2FPT0WU
ALL-PO MV;Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NXXLWod5UUN3ME20O{4{PzlzIN88US=> NGnpT45USU6JRWK=
NCI-H510A MWXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MUHJR|UxRTR5LkmwN|Qh|ryP M1LyN3NCVkeHUh?=

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


+ Expand
  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents individually and in order:
0.5% methylcellulose+0.2% Tween 80
5 mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
in solvent
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID