Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 39 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

Choose Selective PARP Inhibitors

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2. For more details, such as half maximal inhibitory concentrations (IC50s) and working concentrations of each inhibitor, please click on the link of the inhibitor of interest.
3. "+" indicates inhibitory effect. Increased inhibition is marked by a higher "+" designation.
4. Orange "√" refers to compounds which do inhibitory effects on the related isoform, but without specific value.

Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
C41 MmjuT4lv[XOnIFHzd4F6 MYCzNEBucW5? NFHCZ41KdmirYnn0bY9vKG:oIGDBVnAyKHerdHigSWM2OCCxZjCwMlAxOiEQvF2= MWixPVg5QDd4MB?=
Jurkat NUDjclhpU2mwYYPlJGF{e2G7 MX25OkBp NGDwT|RFVVOR MW\Jcohq[mm2aX;uJI9nKFCDUmCxJIF{e2W|c3XkJIF{KHKnZIXjeIlwdiCxZjDj[YxtKH[rYXLpcIl1gSC5aYToJGVEPTBib3[gN{DPxE1? M4HkelI{QDVyMUm5
Capan1 MWXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MWC3NkBp NVv5NWNHTE2VTx?= Mo\mRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDCVmNCOiCpZX7lJI12fGG2ZXSgbJVu[W5iQ3HwZY4yKGOnbHzzJJdqfGhiSVO1NEBw\iB|OT63JO69VQ>? MX2yOFM6QDN6Mx?=
DT40 NGmyO4REgXSxdH;4bYMhSXO|YYm= NEXXVY84OiCq NVnK[IxnTE2VTx?= NWn3UJlzS3m2b4TvfIlkcXS7IHHnZYlve3RiY3jpZ4tmdiCEUlPBNk1l\W[rY3nlcpQhTFR2MDDj[Yxtew>? M4PPR|I1QTJ{NUi3
ML-1 MXvBdI9xfG:2aXOgRZN{[Xl? NVPMNm97Oi53IN88US=> MWGyOEBp NEjUc5hFVVOR M{nUeXN6dmW{Z3nzeIlk[WyueTDlcohidmOnczDUVmFKVC2rbnT1Z4VlKGGyb4D0c5NqeyCrbjDNUE0yKGOnbHzz M2Ozd|I1QDl3MUO1
HCT-116 NWDvSpI1U2mwYYPlJGF{e2G7 M2nlfVAvPSEQvF2= MWSyOEBp NVjxem9qWEGUUDDhZ5Rqfmm2eTDk[YNz\WG|ZYO= M3nwU|I{ODV2MkGz
FaDu MWHDfZRwfG:6aXOgRZN{[Xl? NHroRoUyOCEQvF2= MV[yOEBp M4PETXJm\HWlZYOgeIhmKGOnbHygeoli[mmuaYT5 NIXvbVczOTlzMk[yNC=>
PC-3 NHOxb4ZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MVWxNEDPxE1? M{HIVmlv\HWlZYOgZUB{cWewaX\pZ4FvfCCrbnjpZol1cW:wIHnuJINwdG:weTDmc5Ju[XSrb39CpC=> MnXqNlE2PzF7MUK=
EoL-1-cell M3jpNWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MVXJR|UxRTFwMEe5PEDPxE1? NG\pTZhUSU6JRWK=
NCI-SNU-5 MlnKS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4fXOWlEPTB;Mz6xNlg1OSEQvF2= MU\TRW5ITVJ?
BV-173 M{DBRWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M4XiU2lEPTB;NT60OVQxQSEQvF2= NFznN49USU6JRWK=
HCC1806 M{fYN2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M3;O[2lEPTB;NT63OVE4OyEQvF2= Mn7tV2FPT0WU
HCC2218 NELOeXNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NXzzcI44UUN3ME23Mlc6PzB2IN88US=> NUS2fm9IW0GQR1XS
NCI-H720 MY\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MoezTWM2OD16LkSzOlA{KM7:TR?= NWC3W2M2W0GQR1XS
KASUMI-1 NYXuOHFYT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MXvJR|UxRThwOEmyOlYh|ryP M1nZbnNCVkeHUh?=
CAL-33 NHPaW|VIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MWjJR|UxRTFyLkSzOEDPxE1? NXm0S416W0GQR1XS
Ramos-2G6-4C10 NIn0eW1Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MoLjTWM2OD1zMj60O|UzKM7:TR?= M2Lj[3NCVkeHUh?=
KY821 NUPpW|N7T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MkfaTWM2OD1zMj60PFUh|ryP NIDjSYZUSU6JRWK=
SK-NEP-1 Ml\yS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NY\FcYlGUUN3ME2xN{4yPjZizszN NFz2cnJUSU6JRWK=
MN-60 NXzCc41HT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MXfJR|UxRTF|LkWzPFkh|ryP NGPobnpUSU6JRWK=
DU-145 MkO5S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NXH4VYFqUUN3ME2xN{46ODV|IN88US=> MmLaV2FPT0WU
OS-RC-2 M4n6XGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NEjh[ZFKSzVyPUG1Mlk2QDlizszN NEHYU5hUSU6JRWK=
RPMI-8226 NXn3dmQxT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MVnJR|UxRTF4LkKwOFIh|ryP Mmr1V2FPT0WU
ChaGo-K-1 MmHBS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NEXkdXhKSzVyPUG2MlU{OjVizszN NV3WN2JVW0GQR1XS
GP5d NGGzWGNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MnfyTWM2OD1zNz6wOVMh|ryP Ml7TV2FPT0WU
COLO-668 MnuwS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MlXiTWM2OD1zNz62Nlk1KM7:TR?= NHnxO|FUSU6JRWK=
KYSE-150 NF3IPHhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MmfkTWM2OD1zOD65PVg3KM7:TR?= NIrWRoJUSU6JRWK=
Daoy MUPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mnr3TWM2OD1zOT61OlQ6KM7:TR?= NWqydJk1W0GQR1XS
ECC10 MXPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mn;oTWM2OD1{MD63OFU2KM7:TR?= NXLrZmViW0GQR1XS
A388 M4jKWmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NWC0dXRyUUN3ME2yNU46ODlzIN88US=> MVfTRW5ITVJ?
MHH-NB-11 Mm\HS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NIOyTWhKSzVyPUKzMlE{PjNizszN NIe4eIZUSU6JRWK=
HCC1937 NX7zU3hGT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MVPJR|UxRTJ2Lke0OkDPxE1? MXzTRW5ITVJ?
TGBC11TKB MWnHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MXvJR|UxRTJ3Lk[4OlMh|ryP MnG3V2FPT0WU
CTV-1 Mm\uS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NE[yUI9KSzVyPUK1Mlg6PjlizszN NX\3epZoW0GQR1XS
NCI-H2029 M{HhS2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MmjuTWM2OD1{Nj60NlM5KM7:TR?= M{fqbHNCVkeHUh?=
NCI-H1693 NGroZnhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MmXBTWM2OD1{Nz6yPFk5KM7:TR?= M3P1THNCVkeHUh?=
HCC70 NIDxdoNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M4\ue2lEPTB;MkeuO|I1PiEQvF2= MXvTRW5ITVJ?
BEN MXjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mmj5TWM2OD1{Nz65OVY3KM7:TR?= MkOwV2FPT0WU
LB771 NHjt[5NIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NXjLcFQ2UUN3ME2yPE45Ozd|IN88US=> NHH2[|RUSU6JRWK=
697 MYHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M{PVUWlEPTB;MkmuNFI{PSEQvF2= NInjT25USU6JRWK=
LU-139 MnT1S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M{XhZmlEPTB;MkmuN|c1QCEQvF2= MUjTRW5ITVJ?
EW-13 NFmwXllIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MnXzTWM2OD1{OT6zPFE1KM7:TR?= MkDIV2FPT0WU
MOLT-13 M3P2Rmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NYjVTlR6UUN3ME2yPU4{QDF2IN88US=> M2jINHNCVkeHUh?=
L-363 NYnOU|c2T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NGX4WpdKSzVyPUK5MlQ4QThizszN NGL1R4dUSU6JRWK=
RS4-11 M{PiSGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M4\lWmlEPTB;M{CuOFI1OSEQvF2= M{[4c3NCVkeHUh?=
A2780 NFTSOHdIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MYnJR|UxRTNyLke0OVch|ryP NHi1fGlUSU6JRWK=
KU812 M2e5cmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M4D1XWlEPTB;M{KuN|Y1OiEQvF2= MkLnV2FPT0WU
COLO-684 MVzHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mk\1TWM2OD1|Mz6zOVk6KM7:TR?= MXjTRW5ITVJ?
MFE-280 MWrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MnPxTWM2OD1|Mz6zPFg6KM7:TR?= NGTUUHBUSU6JRWK=
KG-1 NHizNpRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NIrxNm5KSzVyPUOzMlYxODFizszN NXnCbVZ[W0GQR1XS
JVM-3 NE\4ZndIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MVTJR|UxRTN3LkW4Olgh|ryP NFnGZ2JUSU6JRWK=
LAMA-84 MmqwS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? Mkn3TWM2OD1|Nj63N|Q2KM7:TR?= MmrkV2FPT0WU
MOLT-16 NXK0UJdwT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NGTpSWlKSzVyPUO2Mlk2OiEQvF2= M4XuZXNCVkeHUh?=
H4 NFPEPYlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M132NmlEPTB;M{euOVY4KM7:TR?= NVW1eI94W0GQR1XS
T47D NHrEdItIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M33RWGlEPTB;M{euO|AyQCEQvF2= Ml3rV2FPT0WU
CAL-54 NIjmd4lIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NYOwN|dSUUN3ME2zO{46PjZizszN MWHTRW5ITVJ?
SW982 NH30NXBIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MnL6TWM2OD1|OD6wPVk5KM7:TR?= M2\FcHNCVkeHUh?=
IGROV-1 M2DIeGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NEXRdFVKSzVyPUO5MlM{ODRizszN NU\aVnhXW0GQR1XS
KARPAS-45 M{jzXWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NGH2cZNKSzVyPUSxMlQ5OThizszN MkPUV2FPT0WU
JVM-2 MlHzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NUnncpVRUUN3ME20Nk46OjB5IN88US=> Mn73V2FPT0WU
A4-Fuk M1T2eGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NV:0eoZ1UUN3ME20N{42PjlzIN88US=> M{TZdXNCVkeHUh?=
MDA-MB-361 MWDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NGrXPYdKSzVyPUSzMlg1OTRizszN NYjOTWpbW0GQR1XS
T98G M{[5[mdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MlfWTWM2OD12ND64OVE4KM7:TR?= NHLnd|lUSU6JRWK=
Mo-T MkTxS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NVfCeHJwUUN3ME20OU43Ozh7IN88US=> NEfFcJpUSU6JRWK=
ALL-PO NEjQTnRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MoLJTWM2OD12Nz6zO|kyKM7:TR?= NVPsdWExW0GQR1XS
NCI-H510A MlHxS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M1LuTWlEPTB;NEeuPVA{PCEQvF2= M{DUV3NCVkeHUh?=
ML-2 NGDuUmpIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MmPpTWM2OD12OT63PFU3KM7:TR?= MkDSV2FPT0WU

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research
+ Expand
  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water <1 mg/mL
Ethanol <1 mg/mL
In vivo 0.5% methylcellulose+0.2% Tween 80 5 mg/mL

* 1 mg/ml means slightly soluble or insoluble.
* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
in solvent
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT02849496 Not yet recruiting BRCA1 Gene Mutation|BRCA2 Gene Mutation|Estrogen Receptor Negative|HER2/Neu Negative|Progesterone Receptor Negative|Stage IIIA Breast Cancer|Stage IIIB Breast Cancer|Stage IIIC Breast Cancer|Stage IV Breast Cancer|Triple-Negative Breast Carcinoma National Cancer Institute (NCI) May 2017 Phase 2
NCT02831179 Not yet recruiting Functional Pancreatic Neuroendocrine Tumor|Malignant Somatostatinoma|Merkel Cell Carcinoma|Metastatic Adrenal Gland Pheochromocytoma|Metastatic Carcinoid Tumor|Multiple Endocrine Neoplasia Type 1|Multiple Endocrine Neoplasia Type 2A|Multiple Endocrine Neoplasia Type 2B|Neuroendocrine Neoplasm|Non-Functional Pancreatic Neuroendocrine Tumor|Pancreatic Glucagonoma|Pancreatic Insulinoma|Recurrent Adrenal Cortex Carcinoma|Recurrent Adrenal Gland Pheochromocytoma|Recurrent Merkel Cell Carcinoma|Somatostatin-Producing Neuroendocrine Tumor|Stage III Adrenal Cortex Carcinoma|Stage III Thyroid Gland Medullary Carcinoma|Stage IIIA Merkel Cell Carcinoma|Stage IIIB Merkel Cell Carcinoma|Stage IV Adrenal Cortex Carcinoma|Stage IV Merkel Cell Carcinoma|Stage IVA Thyroid Gland Medullary Carcinoma|Stage IVB Thyroid Gland Medullary Carcinoma|Stage IVC Thyroid Gland Medullary Carcinoma|Thymic Carcinoid Tumor|VIP-Producing Neuroendocrine Tumor|Well Differentiated Adrenal Cortex Carcinoma|Zollinger Ellison Syndrome Vanderbilt-Ingram Cancer Center|National Cancer Institute (NCI) August 2016 Phase 1
NCT02631733 Recruiting Estrogen Receptor Negative|HER2/Neu Negative|Neuroendocrine Neoplasm|Progesterone Receptor Negative|Stage IIB Cervical Cancer|Stage IIIA Cervical Cancer|Stage IIIB Cervical Cancer|Stage IIIB Non-Small Cell Lung Cancer|Stage IIIC Breast Cancer|Stage IV Breast Cancer|Stage IV Cervical Cancer|Stage IV Gastric Cancer|Stage IV Non-Small Cell Lung Cancer|Stage IV Ovarian Cancer|Stage IV Small Cell Lung Carcinoma|Triple-Negative Breast Carcinoma National Cancer Institute (NCI) July 2016 Phase 1
NCT02595905 Recruiting BRCA1 Mutation Carrier|BRCA2 Mutation Carrier|Estrogen Receptor Negative|HER2/Neu Negative|Progesterone Receptor Negative|Stage IV Breast Cancer|Triple-Negative Breast Carcinoma National Cancer Institute (NCI) July 2016 Phase 2
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 2016 Phase 1
NCT02483104 Active, not recruiting Ovarian Cancer AbbVie July 2015 Phase 1

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Handling Instructions

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID