Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

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2. For more details, such as half maximal inhibitory concentrations (IC50s) and working concentrations of each inhibitor, please click on the link of the inhibitor of interest.
3. "+" indicates inhibitory effect. Increased inhibition is marked by a higher "+" designation.
4. Orange "√" refers to compounds which do inhibitory effects on the related isoform, but without specific value.

Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
Jurkat MULLbY5ie2ViQYPzZZk> M3HTNFk3KGh? NHjX[WdFVVOR NV7ROWJyUW6qaXLpeIlwdiCxZjDQRXJROSCjc4Pld5Nm\CCjczDy[YR2[3Srb36gc4Yh[2WubDD2bYFjcWyrdImge4l1cCCHQ{WwJI9nKDNizszN MX[yN|g2ODF7OR?=
Capan1 MlTZS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MV23NkBp M4TLN2ROW09? NIjrNG1CdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JGJTS0F{IHflcoUhdXW2YYTl[EBpfW2jbjDDZZBidjFiY3XscJMhf2m2aDDJR|UxKG:oIEO5Mlch|ryP MVuyOFM6QDN6Mx?=
DT40 M4S2bmN6fG:2b4jpZ{BCe3OjeR?= NWjreJVKPzJiaB?= MWfEUXNQ M2TWXWN6fG:2b4jpZ4l1gSCjZ3HpcpN1KGOqaXPr[Y4hSlKFQUKt[IVncWOrZX70JGRVPDBiY3XscJM> MUWyOFkzOjV6Nx?=
ML-1 NY[1dIlMSXCxcITveIlkKEG|c3H5 MnTFNk42KM7:TR?= M3u2blI1KGh? MoDsSG1UVw>? MVTTfY5memerc4TpZ4FtdHliZX7oZY5k\XNiVGLBTWwucW6mdXPl[EBieG:ydH;zbZMhcW5iTVytNUBk\Wyucx?= NHLFe|QzPDh7NUGzOS=>
UM-SCC1 NF;yW3NEgXSxdH;4bYMhSXO|YYm= NIXhZ4IyOCEQvF2= NUnofIRYOjRiaB?= M336NnJm\HWlZYOgeIhmKGOnbHygeoli[mmuaYT5 MWKyNVkyOjZ{MB?=
FaDu M4PBW2N6fG:2b4jpZ{BCe3OjeR?= M3X5O|ExKM7:TR?= M37CcVI1KGh? MmCxVoVlfWOnczD0bIUh[2WubDD2bYFjcWyrdIm= MWOyNVkyOjZ{MB?=
PC-3 MUDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MYGxNEDPxE1? NH\VXFNKdmS3Y3XzJIEhe2mpbnnmbYNidnRiaX7obYJqfGmxbjDpckBkd2yxbomg[o9zdWG2aX;uxsA> M13iXFIyPTdzOUGy
EoL-1-cell NXjqZ49ST3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NILBUFJKSzVyPUGuNFc6QCEQvF2= MkixV2FPT0WU
BV-173 Mlf6S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MYjJR|UxRTVwNEW0NFkh|ryP MW\TRW5ITVJ?
HCC1806 NVGzPY9lT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NX\5[pJJUUN3ME21Mlc2OTd|IN88US=> MlHUV2FPT0WU
HCC2218 MWHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MVPJR|UxRTdwN{m3NFQh|ryP MlXBV2FPT0WU
SK-MEL-24 NUn6cHRqT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= Mmm3TWM2OD15LkixPVI1KM7:TR?= NXvEWmxHW0GQR1XS
NCI-H720 MnjvS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NFThRm5KSzVyPUiuOFM3ODNizszN Mn:0V2FPT0WU
HAL-01 NF;Zd4VIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MXHJR|UxRTlwOEi2NkDPxE1? NXjG[ncxW0GQR1XS
CAL-33 M3;kV2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1fQN2lEPTB;MUCuOFM1KM7:TR?= NEG4b3ZUSU6JRWK=
SK-MEL-1 M1\LNWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1O5OmlEPTB;MUKuOFY3OyEQvF2= M3PKT3NCVkeHUh?=
Ramos-2G6-4C10 NInkUVVIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MoLZTWM2OD1zMj60O|UzKM7:TR?= M3\jSHNCVkeHUh?=
KY821 NFjlc4ZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MUfJR|UxRTF{LkS4OUDPxE1? MY\TRW5ITVJ?
HEC-1 MlXJS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NHj6d|VKSzVyPUGyMlkyQTZizszN NHzMeoFUSU6JRWK=
SK-NEP-1 NFL4NGdIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NVXPbYxwUUN3ME2xN{4yPjZizszN MkDEV2FPT0WU
MN-60 MonyS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NHrX[I5KSzVyPUGzMlU{QDlizszN M4\mcHNCVkeHUh?=
DU-145 M4HzZmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NVnCcJpVUUN3ME2xN{46ODV|IN88US=> NIDqNnpUSU6JRWK=
EW-3 MlLhS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MXnJR|UxRTF2LkW1OlUh|ryP NHjWTpZUSU6JRWK=
OS-RC-2 MlOxS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NYLE[41qUUN3ME2xOU46PTh7IN88US=> MkTMV2FPT0WU
ChaGo-K-1 NIHCWXRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MVvJR|UxRTF4LkWzNlUh|ryP NGjlN5pUSU6JRWK=
DEL MYXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NX;mWVd1UUN3ME2xOk43PzF5IN88US=> MUTTRW5ITVJ?
GP5d M3HTXGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MXTJR|UxRTF5LkC1N{DPxE1? M2HDZ3NCVkeHUh?=
COLO-668 NHv6XY5Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MYDJR|UxRTF5Lk[yPVQh|ryP NFvre3NUSU6JRWK=
H9 NInRRZNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NH[5colKSzVyPUG4MlI5OzNizszN MnzVV2FPT0WU
NKM-1 M3jYOmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NELlS4dKSzVyPUG4MlUyOTlizszN NY\ndXJMW0GQR1XS
KYSE-150 MWDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MlL3TWM2OD1zOD65PVg3KM7:TR?= M4G2OHNCVkeHUh?=
Daoy NUjwVId7T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NH6yS3dKSzVyPUG5MlU3PDlizszN NFTCbJhUSU6JRWK=
A388 MliyS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MljkTWM2OD1{MT65NFkyKM7:TR?= NXn1PWdwW0GQR1XS
MHH-NB-11 M4DjW2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NInseoJKSzVyPUKzMlE{PjNizszN M2G2NnNCVkeHUh?=
HCC1937 NGH0NGpIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NGr1PGRKSzVyPUK0Mlc1PiEQvF2= NEC5PI9USU6JRWK=
CTV-1 M3zwVmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NVLabWlmUUN3ME2yOU45QTZ7IN88US=> NIfDN5JUSU6JRWK=
NCI-H2029 MkC0S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NUDYc48{UUN3ME2yOk41OjN6IN88US=> NGfvd45USU6JRWK=
HCC70 M2\NSWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NILmeHBKSzVyPUK3MlczPDZizszN MW\TRW5ITVJ?
BEN M3;2U2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NInGeoFKSzVyPUK3Mlk2PjZizszN MlywV2FPT0WU
LB771 NVW3dm03T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MVTJR|UxRTJ6LkizO|Mh|ryP NXfRTFRUW0GQR1XS
697 MorES5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NWPPOWFLUUN3ME2yPU4xOjN3IN88US=> M{Pme3NCVkeHUh?=
MOLT-13 NV7VUHJnT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MnPCTWM2OD1{OT6zPFE1KM7:TR?= M2j1ZnNCVkeHUh?=
L-363 M2rvXmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NYXqcpJbUUN3ME2yPU41Pzl6IN88US=> M1e4ZXNCVkeHUh?=
EM-2 M3HsUGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NWTYZ|hzUUN3ME2yPU41QTBzIN88US=> M17v[nNCVkeHUh?=
RS4-11 MX\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFO3cY9KSzVyPUOwMlQzPDFizszN M4XYfnNCVkeHUh?=
A2780 MmnmS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4TCOGlEPTB;M{CuO|Q2PyEQvF2= NFHnTXJUSU6JRWK=
KU812 MX3Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MmnpTWM2OD1|Mj6zOlQzKM7:TR?= M2r5T3NCVkeHUh?=
COLO-684 MXPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NHnmdJpKSzVyPUOzMlM2QTlizszN MkLuV2FPT0WU
MFE-280 Ml\vS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M3XyVmlEPTB;M{OuN|g5QSEQvF2= MX;TRW5ITVJ?
KG-1 NHe4b2FIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= Mk\3TWM2OD1|Mz62NFAyKM7:TR?= NUDlO3JWW0GQR1XS
MV-4-11 NGS5ZY9Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NXf1[WdNUUN3ME2zOU45PDl7IN88US=> M4rvZXNCVkeHUh?=
MOLT-16 M4jEWWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NWL1XnVFUUN3ME2zOk46PTJizszN M{TRRXNCVkeHUh?=
H4 MVLHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M1L4[mlEPTB;M{euOVY4KM7:TR?= MWLTRW5ITVJ?
T47D MX\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M{HEbGlEPTB;M{euO|AyQCEQvF2= M{O4fXNCVkeHUh?=
SW982 NGGwdFRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NX;FbVRIUUN3ME2zPE4xQTl6IN88US=> MWrTRW5ITVJ?
IGROV-1 MWfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M1XDW2lEPTB;M{muN|MxPCEQvF2= M2rRd3NCVkeHUh?=
HCC1187 MmjzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MXLJR|UxRTRzLkK3O|Eh|ryP MoXtV2FPT0WU
SBC-1 Ml\oS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NF;xbndKSzVyPUSxMlMxPjNizszN MW\TRW5ITVJ?
KARPAS-45 MUTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MluxTWM2OD12MT60PFE5KM7:TR?= M{fOd3NCVkeHUh?=
MOLT-4 MVHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MnvpTWM2OD12Mj6yOVM5KM7:TR?= MljOV2FPT0WU
JVM-2 M4HVdGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M2H2OmlEPTB;NEKuPVIxPyEQvF2= NVTQ[VRqW0GQR1XS
A4-Fuk NH3GV2tIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M2HnZmlEPTB;NEOuOVY6OSEQvF2= M3[4NnNCVkeHUh?=
T98G MXnHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Ml7iTWM2OD12ND64OVE4KM7:TR?= NWjneZhWW0GQR1XS
Mo-T MmD3S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NXT2fZpEUUN3ME20OU43Ozh7IN88US=> MlvGV2FPT0WU
NCI-H510A NIHaXlRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M13QUWlEPTB;NEeuPVA{PCEQvF2= M2H2WXNCVkeHUh?=

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


+ Expand
  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water <1 mg/mL
Ethanol <1 mg/mL
In vivo 0.5% methylcellulose+0.2% Tween 80 5 mg/mL

* 1 mg/ml means slightly soluble or insoluble.
* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
in solvent
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID