Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

Choose Selective PARP Inhibitors

Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
Jurkat M4TnTGtqdmG|ZTDBd5NigQ>? M2Lq[Fk3KGh? Mn7ESG1UVw>? M3y2ZmlvcGmkaYTpc44hd2ZiUFHSVFEh[XO|ZYPz[YQh[XNicnXkeYN1cW:wIH;mJINmdGxidnnhZoltcXS7IIfpeIghTUN3MDDv[kA{KM7:TR?= NXHFN41HOjN6NUCxPVk>
Capan1 M1zISWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NXXrXHBPPzJiaB?= Ml;xSG1UVw>? MlnQRY51cXC{b3zp[oVz[XSrdnWgZYN1cX[rdImgZYdicW6|dDDCVmNCOiCpZX7lJI12fGG2ZXSgbJVu[W5iQ3HwZY4yKGOnbHzzJJdqfGhiSVO1NEBw\iB|OT63JO69VQ>? M1LJclI1Ozl6M{iz
DT40 NX3wdpZ7S3m2b4TvfIlkKEG|c3H5 M33zTlczKGh? NI[3TYhFVVOR MVPDfZRwfG:6aXPpeJkh[WejaX7zeEBkcGmla3XuJGJTS0F{LXTl[olkcWWwdDDEWFQxKGOnbHzz Ml;3NlQ6OjJ3OEe=
ML-1 NEfyfVVCeG:ydH;0bYMhSXO|YYm= NXfhd4ZXOi53IN88US=> M3zxUlI1KGh? NGPvOZRFVVOR MYDTfY5memerc4TpZ4FtdHliZX7oZY5k\XNiVGLBTWwucW6mdXPl[EBieG:ydH;zbZMhcW5iTVytNUBk\Wyucx?= MXqyOFg6PTF|NR?=
HCT-116 MmDWT4lv[XOnIFHzd4F6 NXH5XYc2OC53IN88US=> MXmyOEBp NH7s[oNRSVKSIHHjeIl3cXS7IHTlZ5Jm[XOncx?= NIe2dYkzOzB3NEKxNy=>
UM-SCC1 MWPDfZRwfG:6aXOgRZN{[Xl? MnfWNVAh|ryP M1W5TlI1KGh? MmDXVoVlfWOnczD0bIUh[2WubDD2bYFjcWyrdIm= NYe4PYhHOjF7MUK2NlA>
FaDu Mn;mR5l1d3SxeHnjJGF{e2G7 NW\uSlRvOTBizszN MoP4NlQhcA>? NFq1XI9T\WS3Y3XzJJRp\SClZXzsJJZq[WKrbHn0fS=> MUeyNVkyOjZ{MB?=
PC-3 NWq3WY1zT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NIrFUHkyOCEQvF2= M1XEd2lv\HWlZYOgZUB{cWewaX\pZ4FvfCCrbnjpZol1cW:wIHnuJINwdG:weTDmc5Ju[XSrb39CpC=> NU\xUIhZOjF3N{G5NVI>
EoL-1-cell NGrpVIdIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M{fGemlEPTB;MT6wO|k5KM7:TR?= MXjTRW5ITVJ?
NCI-SNU-5 NXSyWYJDT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M13zcmlEPTB;Mz6xNlg1OSEQvF2= Mn7UV2FPT0WU
BV-173 M3K1[mdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1S0dGlEPTB;NT60OVQxQSEQvF2= MkX3V2FPT0WU
HCC1806 MmPlS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NHXM[2hKSzVyPUWuO|UyPzNizszN NEfrNYZUSU6JRWK=
COLO-680 M2nwc2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1XOSWlEPTB;Nj6yNVQxPiEQvF2= MUPTRW5ITVJ?
HCC2218 NFzoOphIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NVq5fJF3UUN3ME23Mlc6PzB2IN88US=> NYDPeVF7W0GQR1XS
NCI-H720 NIDPWoRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M3G0RWlEPTB;OD60N|YxOyEQvF2= NE\abGpUSU6JRWK=
KASUMI-1 MoGyS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M1zne2lEPTB;OD64PVI3PiEQvF2= NXv4ZoNiW0GQR1XS
Ramos-2G6-4C10 NVz6eYRVT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NWPCfmRLUUN3ME2xNk41PzV{IN88US=> MkOzV2FPT0WU
KY821 MXfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NYjPVoRrUUN3ME2xNk41QDVizszN M3\Zb3NCVkeHUh?=
HEC-1 M3\KOGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NXjWZ4JQUUN3ME2xNk46OTl4IN88US=> NGDkUppUSU6JRWK=
SK-NEP-1 M3vPeWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWHJR|UxRTF|LkG2OkDPxE1? NHfsR|RUSU6JRWK=
MN-60 NW[2UIdMT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NXHm[VRuUUN3ME2xN{42Ozh7IN88US=> NYPwTpFCW0GQR1XS
DU-145 NHnZeFFIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MmP5TWM2OD1zMz65NFU{KM7:TR?= NXXrO2N[W0GQR1XS
OS-RC-2 Mlf5S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MYDJR|UxRTF3Lkm1PFkh|ryP Mk\IV2FPT0WU
ChaGo-K-1 MknqS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M3fxbWlEPTB;MU[uOVMzPSEQvF2= MW\TRW5ITVJ?
DEL MlXES5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NY\wTodwUUN3ME2xOk43PzF5IN88US=> M13JbnNCVkeHUh?=
GP5d MV3Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MVXJR|UxRTF5LkC1N{DPxE1? MlX0V2FPT0WU
COLO-668 MlnrS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NGXzSGhKSzVyPUG3MlYzQTRizszN NE\tfGNUSU6JRWK=
H9 MV3Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MW\JR|UxRTF6LkK4N|Mh|ryP NF7pRW5USU6JRWK=
NKM-1 Mmn6S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? Ml\HTWM2OD1zOD61NVE6KM7:TR?= MlLzV2FPT0WU
KYSE-150 NF7DZmpIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NFr2TZlKSzVyPUG4Mlk6QDZizszN M1HaVHNCVkeHUh?=
Daoy M2XwR2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NH:1WHBKSzVyPUG5MlU3PDlizszN M2nMbnNCVkeHUh?=
ECC10 NIrWcY9Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NV\3dFZIUUN3ME2yNE44PDV3IN88US=> MVvTRW5ITVJ?
A388 NEPEXIpIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M4[wSmlEPTB;MkGuPVA6OSEQvF2= NVvMemhLW0GQR1XS
MHH-NB-11 M1jUdmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MnPlTWM2OD1{Mz6xN|Y{KM7:TR?= NEe4dlhUSU6JRWK=
HCC1937 NXjVc5AyT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NVjF[YhHUUN3ME2yOE44PDZizszN NVPlZldzW0GQR1XS
TGBC11TKB MmrWS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnLUTWM2OD1{NT62PFY{KM7:TR?= M3rSeXNCVkeHUh?=
CTV-1 M1jVWGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 Mkn5TWM2OD1{NT64PVY6KM7:TR?= Mk\5V2FPT0WU
NCI-H2029 NV:wOppTT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MX3JR|UxRTJ4LkSyN|gh|ryP NEjib2ZUSU6JRWK=
HCC70 MUHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4POb2lEPTB;MkeuO|I1PiEQvF2= M4rXO3NCVkeHUh?=
LB771 MXzHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MWDJR|UxRTJ6LkizO|Mh|ryP NInkO2ZUSU6JRWK=
697 MYPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4H6eWlEPTB;MkmuNFI{PSEQvF2= MlPWV2FPT0WU
LU-139 NEmxT5RIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MV;JR|UxRTJ7LkO3OFgh|ryP NH\KOmVUSU6JRWK=
EW-13 M4DnWGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M2H6RWlEPTB;MkmuN|gyPCEQvF2= NGTtNW5USU6JRWK=
MOLT-13 M3TwZWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M3PLZ2lEPTB;MkmuN|gyPCEQvF2= NUPCWHVTW0GQR1XS
L-363 M3XLeGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWrJR|UxRTJ7LkS3PVgh|ryP NGLEOIdUSU6JRWK=
EM-2 M1jX[Gdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NEPve4VKSzVyPUK5MlQ6ODFizszN NFPUfG1USU6JRWK=
RS4-11 MlHhS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M1XiXmlEPTB;M{CuOFI1OSEQvF2= NWfpR|N3W0GQR1XS
A2780 MUjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MVPJR|UxRTNyLke0OVch|ryP NHe5SJlUSU6JRWK=
COLO-684 NFTNWWhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MojOTWM2OD1|Mz6zOVk6KM7:TR?= M1u5cnNCVkeHUh?=
MFE-280 MkPzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4q5UmlEPTB;M{OuN|g5QSEQvF2= NEjLNpNUSU6JRWK=
KG-1 M1e0Zmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MWXJR|UxRTN|Lk[wNFEh|ryP NVTkb2V4W0GQR1XS
JVM-3 MmfaS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NGTQe4JKSzVyPUO1MlU5PjhizszN MlWzV2FPT0WU
MV-4-11 NYjq[Jd3T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NHrYUW5KSzVyPUO1Mlg1QTlizszN M1jzZnNCVkeHUh?=
LAMA-84 NFHod4RIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M1GzZ2lEPTB;M{[uO|M1PSEQvF2= NX;zfVRzW0GQR1XS
MOLT-16 MoTOS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NHfMZ3NKSzVyPUO2Mlk2OiEQvF2= MmfXV2FPT0WU
H4 MmPjS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M1zFOGlEPTB;M{euOVY4KM7:TR?= MmLhV2FPT0WU
T47D MV7Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NEH6UI1KSzVyPUO3MlcxOThizszN MUPTRW5ITVJ?
CAL-54 MX7Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M{nzXWlEPTB;M{euPVY3KM7:TR?= Mn[4V2FPT0WU
SW982 NFWye21Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MlnuTWM2OD1|OD6wPVk5KM7:TR?= MWXTRW5ITVJ?
IGROV-1 M4i4cmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MkW4TWM2OD1|OT6zN|A1KM7:TR?= M37JenNCVkeHUh?=
NB14 M2HGRmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NFTCd3dKSzVyPUSwMlcxOzFizszN MXnTRW5ITVJ?
HCC1187 M2H6emdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MXvJR|UxRTRzLkK3O|Eh|ryP NUTY[2l3W0GQR1XS
SBC-1 MUjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NGXMWFVKSzVyPUSxMlMxPjNizszN M1fqe3NCVkeHUh?=
KARPAS-45 M4L0XGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1vacGlEPTB;NEGuOFgyQCEQvF2= M4W2dHNCVkeHUh?=
A4-Fuk MWDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NUS1U5lYUUN3ME20N{42PjlzIN88US=> NWL6Rnp5W0GQR1XS
T98G NGPlR4ZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M12yemlEPTB;NESuPFUyPyEQvF2= NYLXbWxNW0GQR1XS
Mo-T MoHjS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NIPy[2VKSzVyPUS1MlY{QDlizszN NYD3fZhFW0GQR1XS
NCI-H510A MWXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NV3VdmZ[UUN3ME20O{46ODN2IN88US=> M1TIWXNCVkeHUh?=

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


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  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents individually and in order:
0.5% methylcellulose+0.2% Tween 80
5 mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID