Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

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Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
C41 M3zY[2tqdmG|ZTDBd5NigQ>? NXf6fohvOzBibXnu NUfNbYNHUW6qaXLpeIlwdiCxZjDQRXJROSC5aYToJGVEPTBib3[gNE4xODJizszN MkLsNVk5QDh5NkC=
Jurkat NHv2[WpMcW6jc3WgRZN{[Xl? MXK5OkBp NHzsWZpFVVOR NUH2dJVvUW6qaXLpeIlwdiCxZjDQRXJROSCjc4Pld5Nm\CCjczDy[YR2[3Srb36gc4Yh[2WubDD2bYFjcWyrdImge4l1cCCHQ{WwJI9nKDNizszN MXmyN|g2ODF7OR?=
Capan1 NUjp[JdxT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NV;zd4NPPzJiaB?= Mo\sSG1UVw>? NHHabW9CdnSrcILvcIln\XKjdHn2[UBi[3Srdnn0fUBi\2GrboP0JGJTS0F{IHflcoUhdXW2YYTl[EBpfW2jbjDDZZBidjFiY3XscJMhf2m2aDDJR|UxKG:oIEO5Mlch|ryP MUiyOFM6QDN6Mx?=
DT40 NIrPOGlEgXSxdH;4bYMhSXO|YYm= MUG3NkBp MkjkSG1UVw>? MV;DfZRwfG:6aXPpeJkh[WejaX7zeEBkcGmla3XuJGJTS0F{LXTl[olkcWWwdDDEWFQxKGOnbHzz NYC0WW04OjR7MkK1PFc>
FaDu NUX6VGt{S3m2b4TvfIlkKEG|c3H5 MnnrNVAh|ryP NFnud5AzPCCq MWXS[YR2[2W|IITo[UBk\WyuII\pZYJqdGm2eR?= MX6yNVkyOjZ{MB?=
PC-3 MWTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MXGxNEDPxE1? M3;QN2lv\HWlZYOgZUB{cWewaX\pZ4FvfCCrbnjpZol1cW:wIHnuJINwdG:weTDmc5Ju[XSrb39CpC=> MnzoNlE2PzF7MUK=
EoL-1-cell MmjmS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MYTJR|UxRTFwMEe5PEDPxE1? M2Pr[nNCVkeHUh?=
NCI-SNU-5 M1LTfWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M13KdmlEPTB;Mz6xNlg1OSEQvF2= M4XCV3NCVkeHUh?=
HCC1806 Ml\ZS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NUPoSHNqUUN3ME21Mlc2OTd|IN88US=> NGnzWXFUSU6JRWK=
COLO-680 M3XlS2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MXXJR|UxRTZwMkG0NFYh|ryP NYfzb3E5W0GQR1XS
SK-MEL-24 MUTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M1XqTGlEPTB;Nz64NVkzPCEQvF2= NHryXlVUSU6JRWK=
KASUMI-1 MWPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NX3s[|FkUUN3ME24Mlg6OjZ4IN88US=> MY\TRW5ITVJ?
HAL-01 MVPHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MnrHTWM2OD17Lki4OlIh|ryP MoXSV2FPT0WU
CAL-33 MXLHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MnLzTWM2OD1zMD60N|Qh|ryP M2LsOnNCVkeHUh?=
SK-MEL-1 MnTYS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M3:3OWlEPTB;MUKuOFY3OyEQvF2= M2joRnNCVkeHUh?=
Ramos-2G6-4C10 MlTaS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M37afWlEPTB;MUKuOFc2OiEQvF2= M17zNXNCVkeHUh?=
KY821 MlfiS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NFy5dJhKSzVyPUGyMlQ5PSEQvF2= M2DkfHNCVkeHUh?=
SK-NEP-1 M4nlbmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NXvmXVhSUUN3ME2xN{4yPjZizszN Mn:4V2FPT0WU
DU-145 MmHQS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MmX6TWM2OD1zMz65NFU{KM7:TR?= MYrTRW5ITVJ?
OS-RC-2 M3;z[Gdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NHjxdnBKSzVyPUG1Mlk2QDlizszN Mn\SV2FPT0WU
RPMI-8226 NIPQfIhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NVjBU|JkUUN3ME2xOk4zODR{IN88US=> MmTMV2FPT0WU
ChaGo-K-1 M4H0XWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M3fEPGlEPTB;MU[uOVMzPSEQvF2= NEC5fHJUSU6JRWK=
DEL MVrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MmO0TWM2OD1zNj62O|E4KM7:TR?= MnX2V2FPT0WU
GP5d NGWxTIZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MkjDTWM2OD1zNz6wOVMh|ryP MXLTRW5ITVJ?
COLO-668 M3PofWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYPJR|UxRTF5Lk[yPVQh|ryP NEXKVHNUSU6JRWK=
H9 NETPRYtIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MXXJR|UxRTF6LkK4N|Mh|ryP MknQV2FPT0WU
NKM-1 NHu5WJBIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= Mlf6TWM2OD1zOD61NVE6KM7:TR?= NWr3NYJ2W0GQR1XS
KYSE-150 M{\heGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYTJR|UxRTF6Lkm5PFYh|ryP M3[z[3NCVkeHUh?=
Daoy MYLHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MX7JR|UxRTF7LkW2OFkh|ryP MmnRV2FPT0WU
A388 M3HlXWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NHL0S3BKSzVyPUKxMlkxQTFizszN M1TNcHNCVkeHUh?=
MHH-NB-11 M175Xmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MmPLTWM2OD1{Mz6xN|Y{KM7:TR?= NFe3OphUSU6JRWK=
HCC1937 Mn:5S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnHjTWM2OD1{ND63OFYh|ryP NV2xd2RtW0GQR1XS
TGBC11TKB M2Wwcmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NWn5[mczUUN3ME2yOU43QDZ|IN88US=> NYT0TZo4W0GQR1XS
CTV-1 MlzzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MYrJR|UxRTJ3Lki5Olkh|ryP NV;JSpZ5W0GQR1XS
NCI-H2029 NIDBVItIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MYHJR|UxRTJ4LkSyN|gh|ryP MV\TRW5ITVJ?
HLE MXnHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NXvhXHBkUUN3ME2yO{4xPTRizszN M{D0e3NCVkeHUh?=
NCI-H1693 NXfOPVFyT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NFL6VYNKSzVyPUK3MlI5QThizszN M{[0NnNCVkeHUh?=
HCC70 NXXObWtWT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M4\RdGlEPTB;MkeuO|I1PiEQvF2= NH;y[3JUSU6JRWK=
BEN MmjzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NYHCbFNsUUN3ME2yO{46PTZ4IN88US=> NXiwcZdtW0GQR1XS
LB771 MYjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NYfMfpc{UUN3ME2yPE45Ozd|IN88US=> NW\oWoF{W0GQR1XS
697 MnrSS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MkfVTWM2OD1{OT6wNlM2KM7:TR?= M1rOXHNCVkeHUh?=
LU-139 NGniW3hIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MX3JR|UxRTJ7LkO3OFgh|ryP MUjTRW5ITVJ?
EW-13 MX7Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M4PxNGlEPTB;MkmuN|gyPCEQvF2= MnnnV2FPT0WU
MOLT-13 NInWPVlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NEfnSJNKSzVyPUK5MlM5OTRizszN M4DtV3NCVkeHUh?=
L-363 MnTIS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnXJTWM2OD1{OT60O|k5KM7:TR?= MYDTRW5ITVJ?
EM-2 MoS1S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MmPYTWM2OD1{OT60PVAyKM7:TR?= M4TSbnNCVkeHUh?=
A2780 M3[x[mdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NI\nRY5KSzVyPUOwMlc1PTdizszN NVH3fpVvW0GQR1XS
KU812 M1rjbGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NUfQeJVRUUN3ME2zNk4{PjR{IN88US=> NWDsdJY3W0GQR1XS
COLO-684 M3vSWGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NXThUnp6UUN3ME2zN{4{PTl7IN88US=> Mn[4V2FPT0WU
MFE-280 MkjlS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4HuWGlEPTB;M{OuN|g5QSEQvF2= Mn\jV2FPT0WU
JVM-3 NF\HSnFIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NXzLfow4UUN3ME2zOU42QDZ6IN88US=> NYrE[oN4W0GQR1XS
MV-4-11 M4DSZmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NVfaeph7UUN3ME2zOU45PDl7IN88US=> NV7CWFFLW0GQR1XS
LAMA-84 NEjGR3VIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= Mk\mTWM2OD1|Nj63N|Q2KM7:TR?= MlL2V2FPT0WU
MOLT-16 MmTzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NGrQbJJKSzVyPUO2Mlk2OiEQvF2= NGPuV5hUSU6JRWK=
H4 NVjxWWFbT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MlvaTWM2OD1|Nz61Olch|ryP Ml25V2FPT0WU
T47D MoXwS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MWLJR|UxRTN5LkewNVgh|ryP MWrTRW5ITVJ?
CAL-54 MorHS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVfJR|UxRTN5Lkm2OkDPxE1? NHXjeJZUSU6JRWK=
IGROV-1 M{e1cmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NY\mPZBFUUN3ME2zPU4{OzB2IN88US=> MXXTRW5ITVJ?
NB14 MnXrS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MmTOTWM2OD12MD63NFMyKM7:TR?= NXvuZplSW0GQR1XS
HCC1187 MlezS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVvJR|UxRTRzLkK3O|Eh|ryP M3LH[3NCVkeHUh?=
SBC-1 MkO5S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NWD4d5IxUUN3ME20NU4{ODZ|IN88US=> NVfrO|hEW0GQR1XS
KARPAS-45 MYDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NWHwN2FpUUN3ME20NU41QDF6IN88US=> M{HjOHNCVkeHUh?=
JVM-2 M3rPdGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NFzOR4RKSzVyPUSyMlkzODdizszN MmL3V2FPT0WU
A4-Fuk MXjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFXZT4lKSzVyPUSzMlU3QTFizszN NWns[HhYW0GQR1XS
MDA-MB-361 Mm\TS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MkTnTWM2OD12Mz64OFE1KM7:TR?= MnnqV2FPT0WU
T98G M1XyUmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NGD1VndKSzVyPUS0Mlg2OTdizszN MUPTRW5ITVJ?
Mo-T M2THfmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NG\UOGpKSzVyPUS1MlY{QDlizszN NIjXZ49USU6JRWK=

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


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  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents to the product individually and in order:
0.5% methylcellulose+0.2% Tween 80
For best results, use promptly after mixing.
5 mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID