Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

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Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
C41 MYTLbY5ie2ViQYPzZZk> M3nDWVMxKG2rbh?= MXHJcohq[mm2aX;uJI9nKFCDUmCxJJdqfGhiRVO1NEBw\iByLkCwNkDPxE1? MX6xPVg5QDd4MB?=
Jurkat M3rFNWtqdmG|ZTDBd5NigQ>? NVXrOnFZQTZiaB?= M{\ON2ROW09? NX\jOWhGUW6qaXLpeIlwdiCxZjDQRXJROSCjc4Pld5Nm\CCjczDy[YR2[3Srb36gc4Yh[2WubDD2bYFjcWyrdImge4l1cCCHQ{WwJI9nKDNizszN Mn;KNlM5PTBzOUm=
Capan1 Ml;OS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4\EdFczKGh? M3P3WWROW09? MVXBcpRqeHKxbHnm[ZJifGm4ZTDhZ5Rqfmm2eTDh[4FqdnO2IFLSR2EzKGenbnWgcZV1[XSnZDDoeY1idiCFYYDhclEh[2WubIOge4l1cCCLQ{WwJI9nKDN7Lkeg{txO MVmyOFM6QDN6Mx?=
DT40 NYXkbnFrS3m2b4TvfIlkKEG|c3H5 MWe3NkBp MUTEUXNQ MXHDfZRwfG:6aXPpeJkh[WejaX7zeEBkcGmla3XuJGJTS0F{LXTl[olkcWWwdDDEWFQxKGOnbHzz MU[yOFkzOjV6Nx?=
ML-1 M4H6fmFxd3C2b4TpZ{BCe3OjeR?= NV;5ZWJ6Oi53IN88US=> M{\yUVI1KGh? NXHqTW8yTE2VTx?= NWHLeYlZW3mwZYLnbZN1cWOjbHz5JIVvcGGwY3XzJHRTSUmOLXnu[JVk\WRiYYDvdJRwe2m|IHnuJG1NNTFiY3XscJM> MnvrNlQ5QTVzM{W=
HCT-116 NF\1PFFMcW6jc3WgRZN{[Xl? MXiwMlUh|ryP NUXkRopDOjRiaB?= NFfrUVlRSVKSIHHjeIl3cXS7IHTlZ5Jm[XOncx?= NGjYNokzOzB3NEKxNy=>
UM-SCC1 MWXDfZRwfG:6aXOgRZN{[Xl? NWP3dHNpOTBizszN NF30dXEzPCCq M{C0dHJm\HWlZYOgeIhmKGOnbHygeoli[mmuaYT5 Mn36NlE6OTJ4MkC=
FaDu MoPaR5l1d3SxeHnjJGF{e2G7 M4jzTVExKM7:TR?= Mn\oNlQhcA>? M33Gb3Jm\HWlZYOgeIhmKGOnbHygeoli[mmuaYT5 M2PmZ|IyQTF{NkKw
PC-3 M4TmfWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NWrXcXVGOTBizszN NEDGZ|lKdmS3Y3XzJIEhe2mpbnnmbYNidnRiaX7obYJqfGmxbjDpckBkd2yxbomg[o9zdWG2aX;uxsA> M1XtWFIyPTdzOUGy
EoL-1-cell NULCfFJuT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NVPwPIoxUUN3ME2xMlA4QThizszN M3LPbnNCVkeHUh?=
BV-173 Mn7JS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MljkTWM2OD13LkS1OFA6KM7:TR?= NUf6NINnW0GQR1XS
HCC1806 NFzwfnBIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NGjjRYNKSzVyPUWuO|UyPzNizszN MmLnV2FPT0WU
COLO-680 NGL0NVdIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NHrHTmFKSzVyPU[uNlE1ODZizszN MlLvV2FPT0WU
HCC2218 MYfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MXnJR|UxRTdwN{m3NFQh|ryP MYLTRW5ITVJ?
SK-MEL-24 NYPsVJc3T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NV[xfYhzUUN3ME23MlgyQTJ2IN88US=> NVXu[I9JW0GQR1XS
NCI-H720 NWqxZ2I3T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MVrJR|UxRThwNEO2NFMh|ryP M2mzTHNCVkeHUh?=
KASUMI-1 MXjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NF35TYhKSzVyPUiuPFkzPjZizszN NHjMcHNUSU6JRWK=
HAL-01 MnrOS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVzJR|UxRTlwOEi2NkDPxE1? NVHIXlNvW0GQR1XS
SK-MEL-1 MWDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NEPJenBKSzVyPUGyMlQ3PjNizszN MnrhV2FPT0WU
KY821 MmLWS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M{fCN2lEPTB;MUKuOFg2KM7:TR?= MljIV2FPT0WU
HEC-1 MV\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M1XKV2lEPTB;MUKuPVE6PiEQvF2= MlrxV2FPT0WU
SK-NEP-1 MYXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MmX4TWM2OD1zMz6xOlYh|ryP NHnZW2FUSU6JRWK=
MN-60 MmPXS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MVjJR|UxRTF|LkWzPFkh|ryP MVvTRW5ITVJ?
DU-145 Mo\2S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnTiTWM2OD1zMz65NFU{KM7:TR?= NIfBNWtUSU6JRWK=
EW-3 MnHlS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MoLaTWM2OD1zND61OVY2KM7:TR?= MnfjV2FPT0WU
OS-RC-2 NH\nWI9Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MUTJR|UxRTF3Lkm1PFkh|ryP Mln3V2FPT0WU
RPMI-8226 MV3Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MW\JR|UxRTF4LkKwOFIh|ryP NHrrepBUSU6JRWK=
ChaGo-K-1 MVvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NIHoc3JKSzVyPUG2MlU{OjVizszN NH;0UlVUSU6JRWK=
GP5d NF:3dFRIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NF\wXWRKSzVyPUG3MlA2OyEQvF2= NXrn[nVKW0GQR1XS
H9 MnXBS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MnfLTWM2OD1zOD6yPFM{KM7:TR?= M4XrNnNCVkeHUh?=
KYSE-150 M1n1WWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYPJR|UxRTF6Lkm5PFYh|ryP NHG0[WlUSU6JRWK=
Daoy NWGzc|AzT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NFeyepNKSzVyPUG5MlU3PDlizszN NFLBVo9USU6JRWK=
ECC10 NX\STJNpT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= Ml7XTWM2OD1{MD63OFU2KM7:TR?= M2fB[HNCVkeHUh?=
A388 NXnTenl7T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= Mor2TWM2OD1{MT65NFkyKM7:TR?= NYP6Zo5tW0GQR1XS
MHH-NB-11 M2XlTmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M3PlOWlEPTB;MkOuNVM3OyEQvF2= M1\ETnNCVkeHUh?=
HCC1937 NGLTS5ZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NWLleVUzUUN3ME2yOE44PDZizszN NHfye4hUSU6JRWK=
TGBC11TKB M3TnXWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MnXNTWM2OD1{NT62PFY{KM7:TR?= M3SydXNCVkeHUh?=
CTV-1 NYTsdXRzT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NH7CdI9KSzVyPUK1Mlg6PjlizszN M2fEb3NCVkeHUh?=
NCI-H2029 Mlu3S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M4WwSWlEPTB;Mk[uOFI{QCEQvF2= NU\Ye|JjW0GQR1XS
NCI-H1693 M4HNeWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MX\JR|UxRTJ5LkK4PVgh|ryP MYXTRW5ITVJ?
HCC70 MkfZS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M1fwNGlEPTB;MkeuO|I1PiEQvF2= MlTwV2FPT0WU
LB771 MX7Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NF\zO|VKSzVyPUK4Mlg{PzNizszN Mn7uV2FPT0WU
697 NHjQfW9Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NHHMS4RKSzVyPUK5MlAzOzVizszN NEDzXo5USU6JRWK=
LU-139 MYTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mme4TWM2OD1{OT6zO|Q5KM7:TR?= MUnTRW5ITVJ?
EW-13 NWmy[ppLT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M{PUNGlEPTB;MkmuN|gyPCEQvF2= MX;TRW5ITVJ?
L-363 M37GSmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NGLGWnJKSzVyPUK5MlQ4QThizszN MV7TRW5ITVJ?
EM-2 MXXHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MUTJR|UxRTJ7LkS5NFEh|ryP NUnpdY5PW0GQR1XS
RS4-11 M3zQSWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NUj4OlRmUUN3ME2zNE41OjRzIN88US=> MVnTRW5ITVJ?
A2780 MYrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFTvb5BKSzVyPUOwMlc1PTdizszN MlHQV2FPT0WU
KU812 NIDmUpJIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NH\jemFKSzVyPUOyMlM3PDJizszN MlizV2FPT0WU
COLO-684 M370Smdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1fJWWlEPTB;M{OuN|U6QSEQvF2= MWrTRW5ITVJ?
MFE-280 NELCdI1Iem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NVzJd2xjUUN3ME2zN{4{QDh7IN88US=> M4ruenNCVkeHUh?=
KG-1 NHG0PWhIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M3vEdmlEPTB;M{OuOlAxOSEQvF2= Ml60V2FPT0WU
JVM-3 MUHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? Mn3rTWM2OD1|NT61PFY5KM7:TR?= MUjTRW5ITVJ?
MV-4-11 NIe1UIlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M2DDNWlEPTB;M{WuPFQ6QSEQvF2= M3j0[HNCVkeHUh?=
LAMA-84 NUTpdZlsT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NEfwS|NKSzVyPUO2Mlc{PDVizszN NYOxUWtwW0GQR1XS
MOLT-16 MVLHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M2PpfWlEPTB;M{[uPVUzKM7:TR?= NX\WT4lRW0GQR1XS
H4 M1H3d2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYnJR|UxRTN5LkW2O{DPxE1? M4XFXnNCVkeHUh?=
T47D MoHxS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MUPJR|UxRTN5LkewNVgh|ryP M3rJb3NCVkeHUh?=
SW982 NYnndoNbT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NHn4N3RKSzVyPUO4MlA6QThizszN MUjTRW5ITVJ?
HCC1187 MnO1S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MWPJR|UxRTRzLkK3O|Eh|ryP NYrWXo9rW0GQR1XS
SBC-1 NHvwWVlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M1;pfWlEPTB;NEGuN|A3OyEQvF2= M{TGN3NCVkeHUh?=
KARPAS-45 MlfRS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NVexN|NiUUN3ME20NU41QDF6IN88US=> M3Hpc3NCVkeHUh?=
JVM-2 MYTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MljJTWM2OD12Mj65NlA4KM7:TR?= MnrpV2FPT0WU
A4-Fuk M13YW2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M4jCcGlEPTB;NEOuOVY6OSEQvF2= MoK2V2FPT0WU
T98G MmfnS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M{n5SGlEPTB;NESuPFUyPyEQvF2= M4fIfXNCVkeHUh?=
Mo-T MYDHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MoLqTWM2OD12NT62N|g6KM7:TR?= Ml\PV2FPT0WU
MHH-PREB-1 M17rbmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MYPJR|UxRTR3Lke1PFUh|ryP MUDTRW5ITVJ?
NCI-H510A M1S3Nmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MXjJR|UxRTR5LkmwN|Qh|ryP MlnrV2FPT0WU

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


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  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water Insoluble
Ethanol Insoluble
In vivo Add solvents to the product individually and in order:
0.5% methylcellulose+0.2% Tween 80
For best results, use promptly after mixing.
5 mg/mL

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID