Veliparib (ABT-888)

Catalog No.S1004 Synonyms: NSC 737664

Veliparib (ABT-888) Chemical Structure

Molecular Weight(MW): 244.29

Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.

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In DMSO USD 156 In stock
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USD 370 In stock

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Cited by 40 Publications

9 Customer Reviews

  • (A) OVCAR-8 cells were exposed to the indicated concentrations of FdUrd along with vehicle, 3 μM ABT-888 or 300 nM AZD2281 for 24 h. Following washing, ABT-888 and AZD2281 were re-added to the plates initially exposed to these agents, and cells were cultured in the continued presence of ABT-888 and AZD2281 for 8 d until colonies formed. (B) OVCAR-8 cells were exposed continuously to the indicated agents for 8 d. (C) OVCAR-8 cells treated as in (A) except that the indicated concentrations of ABT-888 were used.

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.

    OVCAR-8 cells were plated, treated with indicated concentrations of FdUrd and 3 μM ABT-888 using the exposure schemes depicted in (C) and assayed for clonogenicity (D).

    Cancer Research, 2011, 71: 4944-4954. Veliparib (ABT-888) purchased from Selleck.


    Number of foci detected using laser confocal microscopy and fluorescent Fluor 647 anti-H2A.X-phosphorylated (Ser139) antibody. Double-stranded breaks (red) are clearly augmented in cells incubated with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281 compared with PBS and 1% dimethyl sulfoxide controls. Image analysis was performed using ImageJ and the ‘analyze particle’ function.

    Nucl Med Commun 2011 32, 1046-1051. Veliparib (ABT-888) purchased from Selleck.

    Logarithmic growth curves of human Burkitt lymphoma cells over 5 days with 500 nmol/l of ABT-888 and AZD-2281 in combination with 0 Gy (a), 4 Gy (b), 8 Gy (c), and 12 Gy (d) of external beam radiation. The maximal relative reduction was 65.5% of viable cells and occurred with AZD-2281 (500 nmol/l) on day 5. DMSO, dimethyl sulfoxide.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

  • Colorimetric poly(ADP-ribose) polymerase (PARP) activity assay showing the relative activity of the PARP-1 enzyme in Raji lymphocyte tumor cells. Results show a highly significant difference in PARP activity in the controls [PBS and dimethyl sulfoxide (DMSO)] compared with 24 h incubation with 500 nmol/l of ABT-888 and 500 nmol/l of AZD-2281. A significant increase in PARP enzyme activity is shown in DMSO-incubated cells compared with PBS control.* P < 0.05.

    Nucl Med Commun 2011 32(11), 1046-51. Veliparib (ABT-888) purchased from Selleck.

    T47D breast cancer cells were pretreated with indicated concentrations of ABT-888



    Dr.Zhang of Tianjin Medical University. Veliparib (ABT-888) purchased from Selleck.

  • in vivo suppression of PAR formation by the PARP inhibitor ABT-888 upon induction of DNA damage

    Primary human lung fibroblast cells (MRC-5) were pre-treated with the indicated concentration of the PARP inhibitor ABT-888 for two hours. Oxidative DNA damage was induced by 500 µM H2O2 for 10 min and cellular PARP activity was measured by immuno-staining of poly(ADP)-ribose (PAR) (right panels). The in vivo effect of PARP inhibition was compared to cells without DNA damage induction and inhibitor (control) and H2O2-treated cells without inhibitor.
    Average nuclear PAR staining intensities of more than 50 cells were statistically analysed by Kruskal-Wallis and the post-hoc Dunn’s Multiple Comparison tests (left panel). Asterisks indicate highly significant (p<1%) differences to H2O2-treated cells without PARP inhibitor. Thick horizontal bars mark medians and error bars the interquartile range.


    David Schrmann from University of Base. Veliparib (ABT-888) purchased from Selleck.

    Caption:  451 Lu is a melanoma cell line with high PARP expression that is resistant to temozolomide.  Treatment with 25 µM ABT-888 greatly increased sensitivity to temozolomide compared to cells without ABT-888 treatment as measured by MTS assay.



    Dr. Steve Reuland from University of Colorado Denver. Veliparib (ABT-888) purchased from Selleck.

  • Effect of ABT-888 on the viability of endometrial cancer cell line Hec50 and Ishikawa and ovarian cancer cell line SKOV3,Caov3 and PA-1 was detected by WST-1 method after 3 days treatment.



    Dr. Xiangbing Meng of University of Iowa. Veliparib (ABT-888) purchased from Selleck.

Purity & Quality Control

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2. For more details, such as half maximal inhibitory concentrations (IC50s) and working concentrations of each inhibitor, please click on the link of the inhibitor of interest.
3. "+" indicates inhibitory effect. Increased inhibition is marked by a higher "+" designation.
4. Orange "√" refers to compounds which do inhibitory effects on the related isoform, but without specific value.

Biological Activity

Description Veliparib (ABT-888) is a potent inhibitor of PARP1 and PARP2 with Ki of 5.2 nM and 2.9 nM in cell-free assays, respectively. It is inactive to SIRT2. Phase 3.
Features Increases the efficacy of common cancer therapies such as radiation and alkylating agents.
PARP2 [1]
(Cell-free assay)
PARP1 [1]
(Cell-free assay)
2.9 nM(Ki) 5.2 nM(Ki)
In vitro

ABT-888 is inactive to SIRT2 (>5 μM). [1] ABT-888 inhibits the PARP activity with EC50 of 2 nM in C41 cells. [2] ABT-888 could decrease the PAR levels in both irradiated and nonirradiated H460 cells. ABT-888 also reduces clonogenic survival and inhibits DNA repair by PARP-1 inhibition in H460 cells. ABT-888 increases apoptosis and autophagy in H460 cells when combination with radiation. [3] ABT-888 also inhibits PARP activity in H1299, DU145 and 22RV1 cells and the inhibition is independent of p53 function. ABT-888 (10 μM) suppresses the surviving fraction (SF) by 43% in the clonogenic H1299 cells. ABT-888 shows effective radiosensitivity in oxic H1299 cells. Furthermore, ABT-888 could attenuate the SF of hypoxic-irradiated cells including H1299, DU145 and 22RV1. [4]

Cell Data
Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID
Jurkat MU\LbY5ie2ViQYPzZZk> MXu5OkBp M3LVXmROW09? MnThTY5pcWKrdHnvckBw\iCSQWLQNUBie3Onc4Pl[EBieyC{ZXT1Z5Rqd25ib3[gZ4VtdCC4aXHibYxqfHlid3n0bEBGSzVyIH;mJFMh|ryP NUP0RWZvOjN6NUCxPVk>
Capan1 NWj1OJZWT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= Ml\FO|IhcA>? M3zt[mROW09? NYrocoxSSW62aYDyc4xq\mW{YYTpeoUh[WO2aY\peJkh[WejaX7zeEBDWkODMjDn[Y5mKG23dHH0[YQhcHWvYX6gR4Fx[W5zIHPlcIx{KHerdHigTWM2OCCxZjCzPU44KM7:TR?= NYKyO3pbOjR|OUizPFM>
DT40 MVfDfZRwfG:6aXOgRZN{[Xl? M17Q[VczKGh? M{XM[WROW09? NV7LeFhLS3m2b4TvfIlkcXS7IHHnZYlve3RiY3jpZ4tmdiCEUlPBNk1l\W[rY3nlcpQhTFR2MDDj[Yxtew>? NFTTenUzPDl{MkW4Oy=>
ML-1 NVzhdIxbSXCxcITveIlkKEG|c3H5 NYrsZml7Oi53IN88US=> NUPJUnhyOjRiaB?= NGr2PWlFVVOR MYXTfY5memerc4TpZ4FtdHliZX7oZY5k\XNiVGLBTWwucW6mdXPl[EBieG:ydH;zbZMhcW5iTVytNUBk\Wyucx?= NHP3RWMzPDh7NUGzOS=>
PC-3 NXvQe5J[T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M1XwV|ExKM7:TR?= M1fKNWlv\HWlZYOgZUB{cWewaX\pZ4FvfCCrbnjpZol1cW:wIHnuJINwdG:weTDmc5Ju[XSrb39CpC=> MnrkNlE2PzF7MUK=
EoL-1-cell NVvl[4NzT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M1PQ[GlEPTB;MT6wO|k5KM7:TR?= MkDXV2FPT0WU
NCI-SNU-5 MmO3S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MUXJR|UxRTNwMUK4OFEh|ryP M3vVd3NCVkeHUh?=
BV-173 M{WyW2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NFy2Z4ZKSzVyPUWuOFU1ODlizszN Mm\GV2FPT0WU
HCC1806 MVfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NWHF[m1KUUN3ME21Mlc2OTd|IN88US=> NHvrdWhUSU6JRWK=
COLO-680 Mm\oS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NFm0ZolKSzVyPU[uNlE1ODZizszN MlzGV2FPT0WU
HCC2218 NXPtVYRQT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= M4DuU2lEPTB;Nz63PVcxPCEQvF2= M3\reXNCVkeHUh?=
NCI-H720 MnrOS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MX3JR|UxRThwNEO2NFMh|ryP Mn7mV2FPT0WU
KASUMI-1 M1rsfGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1TTdGlEPTB;OD64PVI3PiEQvF2= MlnFV2FPT0WU
HAL-01 M1v1dmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 M1\aPGlEPTB;OT64PFYzKM7:TR?= NHHENFVUSU6JRWK=
SK-MEL-1 MoDkS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NHLHWXFKSzVyPUGyMlQ3PjNizszN NX2yfpdkW0GQR1XS
Ramos-2G6-4C10 M3u1Nmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NGC4NJBKSzVyPUGyMlQ4PTJizszN M4PpT3NCVkeHUh?=
KY821 NU\3fldLT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NXq1VFlyUUN3ME2xNk41QDVizszN NVL4ZlFKW0GQR1XS
HEC-1 NH6yb3pIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M4WxbmlEPTB;MUKuPVE6PiEQvF2= MnnjV2FPT0WU
SK-NEP-1 MW\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFfacmpKSzVyPUGzMlE3PiEQvF2= M{fmfHNCVkeHUh?=
MN-60 MYHHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MWPJR|UxRTF|LkWzPFkh|ryP NUHD[|JtW0GQR1XS
DU-145 M2P3N2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MVTJR|UxRTF|LkmwOVMh|ryP NWrLOYF3W0GQR1XS
OS-RC-2 M1nacmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NW\QZ3ltUUN3ME2xOU46PTh7IN88US=> Ml;tV2FPT0WU
RPMI-8226 NH\y[GNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NEfFRohKSzVyPUG2MlIxPDJizszN MUTTRW5ITVJ?
ChaGo-K-1 MWjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MkDVTWM2OD1zNj61N|I2KM7:TR?= MXnTRW5ITVJ?
DEL MVrHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MmKzTWM2OD1zNj62O|E4KM7:TR?= NX7DWpVYW0GQR1XS
GP5d MXvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MVPJR|UxRTF5LkC1N{DPxE1? M4f6[HNCVkeHUh?=
COLO-668 NGf4WWlIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MYjJR|UxRTF5Lk[yPVQh|ryP MlPRV2FPT0WU
H9 MkLuS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MWXJR|UxRTF6LkK4N|Mh|ryP M3L5bXNCVkeHUh?=
KYSE-150 MkH1S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M135bmlEPTB;MUiuPVk5PiEQvF2= NV3Ebml[W0GQR1XS
Daoy MXTHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NX3YUZplUUN3ME2xPU42PjR7IN88US=> MWfTRW5ITVJ?
ECC10 NH\Dc4VIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MnLuTWM2OD1{MD63OFU2KM7:TR?= MmjMV2FPT0WU
MHH-NB-11 NYLFT3M4T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= Mme2TWM2OD1{Mz6xN|Y{KM7:TR?= M1\u[XNCVkeHUh?=
HCC1937 NYnOb|dUT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MYnJR|UxRTJ2Lke0OkDPxE1? M4jLRnNCVkeHUh?=
TGBC11TKB Mn7XS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M{i1e2lEPTB;MkWuOlg3OyEQvF2= NULieGZnW0GQR1XS
CTV-1 MmHLS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? MY\JR|UxRTJ3Lki5Olkh|ryP MXjTRW5ITVJ?
NCI-H2029 MV\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M{SwN2lEPTB;Mk[uOFI{QCEQvF2= NWfl[nkyW0GQR1XS
NCI-H1693 M1;aS2dzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MlPsTWM2OD1{Nz6yPFk5KM7:TR?= NFK1eo5USU6JRWK=
HCC70 Mnq2S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NFq0bGVKSzVyPUK3MlczPDZizszN MU\TRW5ITVJ?
BEN Mm\US5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NVniWZhQUUN3ME2yO{46PTZ4IN88US=> NV6zbG56W0GQR1XS
LB771 NXzUS|VUT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= NH7VTI9KSzVyPUK4Mlg{PzNizszN M1\xdXNCVkeHUh?=
697 MX3Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NFO4N3FKSzVyPUK5MlAzOzVizszN MX3TRW5ITVJ?
LU-139 NFfveWVIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MkfYTWM2OD1{OT6zO|Q5KM7:TR?= NHn6V|dUSU6JRWK=
EW-13 M1vlOGdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MkPrTWM2OD1{OT6zPFE1KM7:TR?= NYXxXoFMW0GQR1XS
MOLT-13 M4OyVmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MofzTWM2OD1{OT6zPFE1KM7:TR?= M3zOW3NCVkeHUh?=
L-363 NYf0U|RsT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MXzJR|UxRTJ7LkS3PVgh|ryP NE\PNVdUSU6JRWK=
EM-2 MkDWS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? M3\LU2lEPTB;MkmuOFkxOSEQvF2= MX\TRW5ITVJ?
RS4-11 MlfzS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NGXvRlBKSzVyPUOwMlQzPDFizszN NYC5NXhUW0GQR1XS
A2780 NHjYZYxIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NX3pUG1jUUN3ME2zNE44PDV5IN88US=> M3vEbXNCVkeHUh?=
KU812 Moj6S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NVq4TXR5UUN3ME2zNk4{PjR{IN88US=> M1PTOnNCVkeHUh?=
MFE-280 NFOwOlFIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= MorRTWM2OD1|Mz6zPFg6KM7:TR?= NYG2TFFDW0GQR1XS
KG-1 MUfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? M1\RR2lEPTB;M{OuOlAxOSEQvF2= M{DkUHNCVkeHUh?=
MV-4-11 M3r1Tmdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 NFvEcm1KSzVyPUO1Mlg1QTlizszN NIexb5lUSU6JRWK=
LAMA-84 MU\Hdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NIqxVlRKSzVyPUO2Mlc{PDVizszN NXzJcpB6W0GQR1XS
MOLT-16 Mnj6S5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? Mmf5TWM2OD1|Nj65OVIh|ryP MoDwV2FPT0WU
H4 MXjHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MnizTWM2OD1|Nz61Olch|ryP NUL3OYNkW0GQR1XS
T47D NVrNN2t3T3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MWDJR|UxRTN5LkewNVgh|ryP NUSzbVF5W0GQR1XS
CAL-54 NF34c|dIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NUnrO5JEUUN3ME2zO{46PjZizszN NVXO[otkW0GQR1XS
SW982 M3T6PWdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MU\JR|UxRTN6LkC5PVgh|ryP NITmSmRUSU6JRWK=
NB14 MVvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NX3BeXdDUUN3ME20NE44ODNzIN88US=> MmnpV2FPT0WU
HCC1187 NWfR[IlmT3Kxd4ToJGlvcGmkaYTpc44hSXO|YYm= MVHJR|UxRTRzLkK3O|Eh|ryP M3LXUnNCVkeHUh?=
KARPAS-45 NGC4fpNIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= M37GS2lEPTB;NEGuOFgyQCEQvF2= M3;LOnNCVkeHUh?=
MOLT-4 MVfHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? MXLJR|UxRTR{LkK1N|gh|ryP NFvKe3lUSU6JRWK=
A4-Fuk NETmUGZIem:5dHigTY5pcWKrdHnvckBCe3OjeR?= NX\OSVRtUUN3ME20N{42PjlzIN88US=> M1XzfnNCVkeHUh?=
MHH-PREB-1 MVvHdo94fGhiSX7obYJqfGmxbjDBd5NigQ>? NIfn[mpKSzVyPUS1Mlc2QDVizszN MYXTRW5ITVJ?
NCI-H510A MkHIS5Jwf3SqIFnubIljcXSrb36gRZN{[Xl? NGfKPZFKSzVyPUS3MlkxOzRizszN M2ruXnNCVkeHUh?=
ML-2 M3q0[mdzd3e2aDDJcohq[mm2aX;uJGF{e2G7 MX7JR|UxRTR7Lke4OVYh|ryP Mn[wV2FPT0WU

... Click to View More Cell Line Experimental Data

In vivo The oral bioavailability of ABT-888 is 56%-92% in mice, Sprague-Dawley rats, beagle dogs, and cynomolgus monkeys after oral administration. [1] ABT-888 (25 mg/kg i.p.) could improve tumor growth delay in a NCI-H460 xenograft model with well tolerated. Combination with radiation, ABT-888 decreases the tumor vessel formation. [3] ABT-888 reduces intratumor PAR levels by more than 95% at a dose of 3 and 12.5 mg/kg in A375 and Colo829 xenograft models and the suppression could be maintained over time. [4]


Animal Research:


+ Expand
  • Animal Models: NCI-H460, H460, B16F10 and 9L xenografts in C57BL/6 mice
  • Formulation: Formulated in solution containing 0.9% NaCl adjusted to pH 4.0
  • Dosages: ~25 mg/kg
  • Administration: Orally administered
    (Only for Reference)

Solubility (25°C)

In vitro DMSO 17 mg/mL (69.58 mM)
Water <1 mg/mL
Ethanol <1 mg/mL
In vivo 0.5% methylcellulose+0.2% Tween 80 5 mg/mL

* 1 mg/ml means slightly soluble or insoluble.
* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 244.29


CAS No. 912444-00-9
Storage powder
in solvent
Synonyms NSC 737664

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT00553189 Completed Solid Tumors|Lymphomas National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) August 9, 2007 Phase 1
NCT03032614 Not yet recruiting Breast Cancer Stage IV|Ovarian Cancer|BRCA1 Mutation|BRCA2 Mutation The University of Texas Health Science Center at San Antonio March 31, 2017 Phase 2
NCT01445522 Completed Neoplasms|Lymphoma National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) December 3, 2008 Phase 1
NCT01419548 Withdrawn Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) July 29, 2011 Phase 1
NCT02723864 Recruiting Neoplasms National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) March 22, 2016 Phase 1
NCT00994071 Completed Medulloblastoma|Pontine Glioma|Ependymoma|Astrocytoma|PNET National Cancer Institute (NCI)|National Institutes of Health Clinical Center (CC) September 22, 2009 Phase 1

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID