Catalog No.S1415 Synonyms: SR-25990C

Clopidogrel Chemical Structure

Molecular Weight(MW): 419.9

Clopidogrel is an oral, thienopyridine class antiplatelet agent.

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1 Customer Review

  • (A) HPLC glycan traces of IgG recovered from joint after K/BxN and PBS or B4ST6Fc treatment with or without clopidogrel. (B) Ratios of monosialylated and agalactosylated glycans (S1/G0) from IgG described in (A).

    Cell, 2018, 172(3):564-577.e13. Clopidogrel purchased from Selleck.

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Biological Activity

Description Clopidogrel is an oral, thienopyridine class antiplatelet agent.
P2Y12 [1]
In vitro

Clopidogrel is converted to its active metabolite by cytochrome P450 (CYP) enzymes. [1] Clopidogrel (1 μM) also inhibits EGF-stimulated EGF receptor, PERK expression, and cell proliferation in RGM-1 cells (P<0.05), and causes much less inhibition of EGF-stimulated cell proliferation in EGF receptor over-expressed RGM-1 cells than in RGM-1 cells (22% vs. 32% reduction). [2] Clopidogrel increases blood vessel number, reduces polymorphonuclear count and decreases attachment and bone loss, also decreases osteoclast number in rats submitted or not to periodontal repair. Clopidogrel decreases CXCL4, CXCL12 and PDGF content compared with saline-treated rats, without affecting CXCL5. [3]

In vivo Clopidogrel (2mg and 10mg/kg/day) significantly decreases ulcer-induced gastric epithelial cell proliferation and ulcer-stimulated expressions of EGF receptor and phosphorylated extracellular signal-regulated kinase (PERK) at the ulcer margin of rats. [2] Clopidogrel improves endothelial function and NO bioavailability in rats with congestive heart failure. Clopidogrel-treated Congestive heart failure (CHF) rat displays enhances phosphorylation of AKT and eNOS. [4] The clopidogrel/aspirin combination shows only additive-type effects on bleeding time prolongation induced by ear transection in the rabbit, therefore showing that combined inhibition of cyclooxygenase and ADP's effects provide a marked enhanced antithrombotic efficacy. [5]


Solubility (25°C)

In vitro DMSO 83 mg/mL (197.66 mM)
Water 78 mg/mL (185.75 mM)
Ethanol 46 mg/mL (109.54 mM)

* Please note that Selleck tests the solubility of all compounds in-house, and the actual solubility may differ slightly from published values. This is normal and is due to slight batch-to-batch variations.

Chemical Information

Molecular Weight 419.9


CAS No. 120202-66-6
Storage powder
in solvent
Synonyms SR-25990C

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Clinical Trial Information

NCT Number Recruitment Conditions Sponsor/Collaborators Start Date Phases
NCT02866175 Not yet recruiting Atrial Fibrillation Daiichi Sankyo Inc.|European Cardiovascular Research Institute|Kompetenznetz Vorhofflimmern e.V.|Chiltern International Inc. February 2017 Phase 3
NCT03023020 Not yet recruiting High Bleeding Risk|Coronary Artery Disease|PCI ECRI bv|Cardialysis B.V.|European Cardiovascular Research Center|University of Bern|Terumo Medical Corporation February 2017 --
NCT03006835 Not yet recruiting Coronary Heart Disease Xijing Hospital|Shenzhen Salubris Pharmaceuticals Co., Ltd. January 2017 Phase 4
NCT02663713 Recruiting Myocardial Infarction|Diabetes Mellitus|Renal Disease|Coronary Artery Disease University of Patras|AstraZeneca January 2017 Phase 4
NCT02677545 Recruiting Carotid Artery Stenosis University Hospital, Basel, Switzerland December 2016 Phase 2
NCT02975076 Not yet recruiting Stroke, Acute|Ischemic Attack, Transient Xinhua Hospital, Shanghai Jiao Tong University School of Medicine December 2016 --

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Cell Lines Assay Type Concentration Incubation Time Formulation Activity Description PMID